hormons. hormones are released from glands and they all belong to the endocrine system.
"REPRODUCTIVE ISOLATION" needs to occur for speciation to happen.....
Only the bases varies from one nucleotide to another.
* More than 40 proteins and glycoproteins involved in the complement system are synthesized by the liver, macrophages, epithelial cells, they are present in the blood in plasmatic form, membrane, some have an enzymatic activity, regulator or membrane receptorThese are elements of the humoral innate immune response, they fight infections, purify immune complexes and apoptotic bodies.
<span>There are indeed three ways to activate the complement:</span>
Classical pathway: Activated by Immunoglobulins in immune complexes, aggregated Immunoglobulins, DNA, CRP, apoptotic bodies .......it involves nine fractions, starting with C1, then C4, C2, C3, to form a classical C5 convertase, then, activation of C5, C6, C7, C8, C9.
Alternative pathway: activated by polysaccharides (bacterial endotoxin), vascular wall poor in sialic acid, aggregated IgE ...C3b like is the first component in the alternate channel cascade, it will create an amplification loop, and form an alternative C5 convertase.
Lecithin pathway: Activated by mannose, fucose (carbohydrate of microorganisms)The first component is the complex MBL / MASP1 / MASP2: "mannose-binding protein": works according to the same principle as the complex C1 of the classical way (MASP2 cleaves the C4 and the rest of the cascade is equivalent to that of the classical way).
the three ways have the same outcome: A C5 convertase (formed by one of the pathways) cleaves C5 into C5a and C5b: C5b is deposited far from other fractions on the antigenic surface. The fixation of C5b in the cell is followed by that of C6, C7, C8, and C9 (9 molecules of C9): formation of the membrane attack complex (MAC) ==> Death of the cell by osmotic shock
The initial stage of atherosclerosis is characterized by accumulation beneath the endothelium of excessive amounts of LDL.
- The accumulation of lipids, cholesterol, and other chemicals in and on the artery walls is known as atherosclerosis. The accumulation is known as plaque.
- The build-up of lipids and fibrous components in the major arteries is a hallmark of the degenerative illness known as atherosclerosis. A picture of a typical artery's anatomy can be found in Foam cells, or subendothelial accumulations of cholesterol-engorged macrophages, are the earliest lesions of atherosclerosis.
- Nitric oxide and prostacyclin are two chemicals that are produced by your endothelium. These maintain the fluid in your blood and stop it from clotting when it shouldn't. These compounds' insufficient production is a symptom of various diseases, which raises your risk of clotting.
To learn more about atherosclerosis.
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