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Harlamova29_29 [7]
3 years ago
5

Explain how thunderstorms form.

Biology
1 answer:
Brums [2.3K]3 years ago
8 0

Answer:

Thunderstorm form when warm,moist air rises into cold air

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Ray Of Light [21]

Answer:

a organ have cell wit different shapes and function

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4 years ago
Imagine that you are performing a cross involving seed color in garden pea plants. What traits would you expect to observe in th
Natali [406]

Answer:

Yellow seed colour (heterozygous)

Explanation:

Since we are performing experiment on the garden pea, so in the garden pea Yellow seed color is dominant and green one is recessive.

The allele responsible for the seed colour is denoted by Y.

So,

Homozygous parent having Yellow seeds will have allelic configuration as YY and the other homozygous parent with green seeds will have yy as pair of alleles.

In the cross of YY × yy the F1 progeny will be Yy which means F1 offspring will will be heterozygous and have Yellow seed colour because Yellow seed colour is dominant over the green colour. Green colour will express itself only in the homozygous condition.

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3 years ago
Carrying capacity is _____.
Dominik [7]
The answer is d ...the number of organisms an ecosystem can support
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What are the benefits of studying biology
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4.6 Fusion of the reading frames of the bcr and abl genes, as is often observed in cases of chronic myelogenous leukemia (CML),
kirill115 [55]

Answer:

The answer is reciprocal chromosomal translocation

Explanation:

The Philadelphia chromosome (Ph) is the truncated chromosome 22 generated by the reciprocal translocation t(9;22)(q34;q11) and was first identified in 1960 in a patient with CML [3]. Translocation of the proto-oncogene tyrosine-protein kinase (ABL1) gene located on chromosome 9 to the breakpoint cluster region (BCR) gene located on chromosome 22 results in a BCR-ABL1 fusion gene on the Ph [4, 5]. Three BCR-ABL1 fusion gene hybrids encode BCR-ABL1 protein isoforms p210, p190, and p230, which have persistently enhanced tyrosine kinase (TK) activity. These aberrantly activated kinases disturb downstream signaling pathways, causing enhanced proliferation, differentiation arrest, and resistance to cell death [6, 7]. Tyrosine kinase inhibitors (TKIs) targeting the BCR-ABL1 protein are the most successful targeted therapy for Ph-positive leukemia.

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