Because Muscles that are inactive have a higher incidence of venous stasis.
Venous stasis can occur when the muscles of the extremities are inactive. Venous stasis is a risk factor in Virchow's triad.
<h3>What Is Venous Thromboembolism?</h3>
Venous thromboembolism (VTE), additionally called blood clots, is a sickness that consists of deep vein thrombosis and pulmonary embolism. A deep vein thrombosis (DVT) takes place while a blood clot bureaucracy in a deep vein, typically withinside the decrease leg, thigh, or pelvis.
Your risk of developing VTE is highest after major surgery or serious injury, or when you have heart failure, cancer, or a heart attack. Swelling, redness, and pain are some of the symptoms of deep vein thrombosis. Pulmonary embolism can cause sudden chest pain and shortness of breath.
VTE sometimes occurs without any obvious signs. Medicines to help prevent more blood clots from forming or to clear up severe vein blockages are the mainstay of treatment for VTE.
To learn more about venous thromboembolism (VTE) from the given link
brainly.com/question/2870265
#SPJ4
Answer:
Bacteria are highly adaptable microorganisms who have the capability of developing defense mechanisms against that which may harm them. Not least important of all, is the easiness with which some bacteria, especially pathogenic bacteria like Salmonella, or Klebsiella, develop mechanisms of resistance to antiseptics and, most importantly, antibiotics.
Antibiotics are a chemical substance that was created, and has been developed, in order to be able to combat pathogenic microorganisms, specifically bacteria. However, because today these substances are being used indiscriminately, we are now seeing a very worrying pattern of antibiotic-resistance patterns in microorganisms that used to be sensible to them. The result, we are facing strains of pathogenic bacteria, like Klebsiella pneumonia and E. Coli, that have become resistan to all types of antibiotics, from first generation, to fourth generation. And this has meant that when people acquire infection by these pathogens, the likelihood of death by them has increased because there are no agents capable of combating them.
Exposure to antibiotics has been the sole reason why these resistant strains of bacteria have emerged, especially when these antibiotics are not necessary. And feeding these substances to animals, to ensure their development and weight gain, has not made the situation any better. Now, we are instead adding also bacteria to the list that did not use to be resistant, but that are becoming so as they become adjusted to the constant exposure to antibiotics. Again, the result has been: more people infected with bacterial strains that cannot be combated with any of the existing antibiotic agents.
Osteoporosis affects the a) skeletal body system
Answer:
Rostral
Explanation:
according to google rostral means: situated or occurring near the front end of the body, especially in the region of the nose and mouth or (in an embryo) near the hypophyseal region
Answer:
b) blastic red blood cell (RBC).
Explanation:
In excess of 340 blood group antigens have now been described that vary between individuals. Thus, any unit of blood that is nonautologous represents a significant dose of alloantigen. Most blood group antigens are proteins, which differ by a single amino acid between donors and recipients. Approximately 1 out of every 70 individuals are transfused each year (in the United States alone), which leads to antibody responses to red blood cell <u>(RBC) alloantigens</u> in some transfusion recipients. When alloantibodies are formed, in many cases, RBCs expressing the antigen in question can no longer be safely transfused. However, despite chronic transfusion, only 3% to 10% of recipients (in general) mount an alloantibody response. In some disease states, rates of alloimmunization are much higher (eg, sickle cell disease). For patients who become alloimmunized to multiple antigens, ongoing transfusion therapy becomes increasingly difficult or, in some cases, impossible. While alloantibodies are the ultimate immune effector of humoral alloimmunization, the cellular underpinnings of the immune system that lead to ultimate alloantibody production are complex, including antigen consumption, antigen processing, antigen presentation, T-cell biology.
