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kirill115 [55]
2 years ago
11

WILL GIVE BRIANLIEST!

Biology
1 answer:
vladimir2022 [97]2 years ago
4 0
We usually view objects when they are illuminated by white light, usually sunlight or ordinary room light. White light is a mixture of all colors, in roughly equal proportions. White objects look white because they reflect back all the visible wavelengths of light that shine on them - so the light still looks white to us. Colored objects, on the other hand, reflect back only some of the wavelengths; the rest they absorb. For example, if white light shines on a red ball, the ball reflects back mostly red light, and so we see red. Most of the greens and blues that are part of white light are absorbed by the ball so we cannot see them. Likewise, a blue book is reflecting the blue part of the white light spectrum. The red and green parts are absorbed by the book.

What happens when red light shines on a red ball? It continues to reflect the red light, and so it is still red -- but a white ball would also look red in red light, because it reflects all colors. If instead we shine blue light on a red ball, it will look dark, because it does not reflect blue light. It cannot look red unless there is red light coming to it from the light source. And it cannot look blue because the red ball absorbs blue light. So when we ask what color an object is, the answer is not simple - it depends on what color light we are using to see the object.

One consequence of the fact that different colored objects absorb different wavelengths of light is that darker objects heat up faster in the sun than white ones do - because they absorb many of the different wavelengths of light energy, while white objects reflect most of the wavelengths.
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2 years ago
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Use the drop down menus to match the following descriptions to the corresponding lymphatic structures.
Reil [10]

Answer:

<em>Well, Cells of the immune system not only use lymphatic vessels to make their way and T cells mature in the thymus, they both initially develop from bone marrow. B cells are immune cells that function primarily by producing antibodies. structure on the surface of a pathogen that binds to T or B lymphocyte antigen receptors. Hope That Helps!</em>

<em>From Itsbrazts.</em>

3 0
3 years ago
One hypothesis that proposes to explain senescence notes that cells that divide throughout the life course have shortened dna se
mylen [45]

Answer:

telomere hypothesis

Explanation:

Telomere hypothesis -

Telomere -

It is the area where there are repetitive nucleotide sequence at the very end of the chromosome that does not allow the ends from fusing with the other chromosomes and even from deteriorating .

<u>according to the hypothesis , the cells have shorter DNA sequence which divide throughout the life .</u>

<u>hence , the correct term for the given statement is - telomere hypothesis  . </u>

6 0
3 years ago
Indicate whether each of the following statements is true of depurination (DP), deamination (DA), or pyrimidine dimer formation
solniwko [45]

Answer:

- This process is caused by spontaneous hydrolysis of a glycosidic bond: depurination and deamination

- This process is induced by ultraviolet light:  pyrimidine dimer formation

- This can happen to guanine but not to cytosine: depurination

- This can happen to thymine but not to adenine:  pyrimidine dimer formation

- This can happen to thymine but not to cytosine: none

- Repair involves a DNA glycosylase: deamination

- Repair involves an endonuclease: depurination, deamination and  pyrimidine dimer formation

- Repair involves DNA ligase: depurination, deamination and  pyrimidine dimer formation

-  Repair depends on the existence of separate copies of the genetic information in the two strands of the double helix: depurination, deamination and  pyrimidine dimer formation

- Repair depends on cleavage of both strands of the double helix: none

Explanation:

Depurination is the loss of purine bases (either adenine or guanine), while deamination refers to the removal of an amino group. During depurination, a β-N-glycosidic bond is cleaved by hydrolysis and a nucleic base is released (either adenine or guanine). All DNA bases may undergo deamination, except thymine (since thymine does not have an amino group). The ultraviolet (UV) radiation can cause thymine or cytosine to form dimers (e.g., pyrimidine dimers), being thymine dimers the most common lesion when DNA is exposed to UV light. Pyrimidine dimers may be repaired by different excision mechanisms, e.g., nucleotide excision repair, where the recognition of the DNA damage leads to the removal of the DNA fragment containing the lesion. DNA glycosylases are enzymes involved in the mechanism of base excision, these enzymes recognize and remove damaged bases by hydrolysis of the glycosidic bond, producing an abasic (apurinic and apyrimidinic) site. A DNA ligase enzyme covalently joins two DNA molecules by forming a phosphodiester bond, which is required during these processes.

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