This is simply a protective mechanism of the body. A person with blood group A will produced antibodies against blood group B because group B cells are considered foreign bodies to the person and will not be tolerated and vice versa. Group B people have antibodies against group A and will not tolerate group A cells. This is why group A person can only be transfused with group A blood or group O blood. Both A and B have no antibodies against blood group O. Indeed no blood group has antibodies against Blood group and this is why people with blood group O are called universal donors.
Group AB people do not have antibodies against any blood group because both these groups are part and parcel of their body and production of antibodies against either A or B would end up in self destruction of the body. Group AB people are known as universal recipients because they can receive blood from any of the other groups.
Answer:
Test crosses are used in selective breeding because they can determine the genotype of the parent.
Explanation:
Also, I see it it ur first question. Welcome to Brainly!
The best explanation for the greater rate of transport for solute A than for solute B at higher solute concentrations is that there are more frequent collisions of molecules of A than that of B.
<h3>How does concentration affect transport of molecules</h3>
- Concentration is the amount of solute dissolved in a given volume of solution.
Given two solutions A and B of different concentrations, The solution of higher concentration will have a higher amount of dissolved solutes.
Given A has a higher concentration, the solutes will collide with themselves much more than in B.
Hence, the molecules of A will tend to move faster than that of B.
Therefore, the best explanation for the greater rate of transport for solute A than for solute B at higher solute concentrations is that there are more frequent collisions of molecules of A than that of B.
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(1) X. Moons shadow creates solar eclipse.
(2) ✓
(3) ✓
(4) covered by the sun
(5)new moon
You would expect to see abnormal limb development.
A long-range shh enhancer regulates expression in the developing limb and fin is associated with preaxial polydactyly. Consequences of deletion and mutation of the limb enhancer in a mouse would be an abnormal development of the limb which is regulated by the Shh enhancer.
