The breakdown of carbohydrates into glucose. Glucose use to obtain energy (ATP) for the human cellular respiration by the steps glycolysis, acetyl CoA and kreb cycle. In glycolysis, ATP is use to generate greater ATP called the ATP debt. Fat (lipid) and protein (amino acid) also can give human energy call gluconeogenesis but only when critical condition only.
Answer:
false
Explanation:
im not sure if this is the entire question but from what I can see, I would prove this false
The formula for aerobic respiration in animals can be shown by the equation below. A glucose molecule is broken down by enzymes in the presence of oxygen to make carbon dioxide, water, and ATPs. Carbon IV oxide is expelled by the lungs.
C₆H₁₂O₆ + 6O₂ --> 6CO₂ + 6H₂O + 38 ATP
This process occurs in two stages. Glycolysis (that does not require oxygen) occurs in the cytoplasm of cells. The products of the glucose breakdown in this stage is pyruvate, CO₂ and 2 net ATPs. The pyruvate is then converted to acetyl-CoA that enters the second stage called Citric/Krebs cycle. This second stage happens in the mitochondria. The products are CO₂, H₂O and 34 ATPs
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Change in velocity, direction, or both
I think that’s what you’re asking
Blood flows through an artery that is partially blocked. as the blood moves from the wider region into the narrow region, alternately increases and then decreases.
<h3>What is the purpose of blood flow in heart?</h3>
The removal of positive charge by electrochemical gradients causes the membrane potential of the cell to shift more strongly in the direction of the equilibrium potential for potassium.
The membrane would become more negative as potassium permeability through the membrane increased. Given that potassium is a positively-charged ion and that the intracellular environment has a net negative charge, enhancing the membrane's permeability would result in a more negative membrane.
For instance, the amplitude of the potassium concentration gradient across the myocyte decreases as the external potassium level rises, lowering the resting membrane potential.
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