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Mazyrski [523]
3 years ago
12

A small group of diploid triangular Sticky Notes were fluttering along the coast of The Mainland. A storm blew in and blew the S

ticky Notes to an isolated island that had never previously been inhabited by Sticky Notes. After a few years, the Sticky Note population (though still small) has happily made the new island their home and are visited by an evolutionary biologist. The evolutionary biologist bands the Notes, collects tissue for a genetic analysis, and finds that this isolated population of Notes is not in Hardy-Weinberg equilibrium. Why is this, and what may be the potential long-term significance
Biology
1 answer:
muminat3 years ago
5 0

Answer:

Because genetic drift (Founder effect) is acting on this population. Not all the Hardy weinberg criteria are accomplished. There are no random matings and populations are finite-sized.

Explanation:

This is a special case of genetic drift: the founder effect.

The “Founder effect” phenomenon refers to cases where a new population originates from a few founder individuals, coming from a bigger ancestral population, that established in a new environment. This small population might or might not be genetically representative of the original one. This subgroup carries with them some genetic information that they share with their original population. Over time, some genes can be lost, or they can increase in frequency. Some rare alleles might be exceeded or might be completely lost. On Consequence, when the small population grows, it will have a genetically different composition from the original one. In these situations, genetic variability is reduced and enhances the possibility of developing a peculiar allelic composition. In some cases, the founder effect is part of the process of speciation.  

The criteria for maintaining a Hardy-Weinberg equilibrium are:  

  • Random matings
  • No superposed generations
  • No mutations
  • No migration  
  • Infinite population size
  • No natural selection

Genetic drift involved the un-accomplishment of random matings and infinite population sizes.

Genetic drift involves:

  • limited population sizes
  • individuals reproduce by endogamy/exogamy, and matings occur by phenotype.

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JulijaS [17]

Answer:

Antibiotics save lives but any time antibiotics are used, they can cause side effects and lead to antibiotic resistance.

Since the 1940s, antibiotics have greatly reduced illness and death from infectious diseases. However, as we use the drugs, germs develop defense strategies against them. This makes the drugs less effective.

6 0
3 years ago
Place all eight of these steps of the insulin signaling pathway in the correct order. The abbreviation PIP 2 is used for phospha
DedPeter [7]

Answer:

The correct insulin pathway is described as under:

2. Binding of insulin to the alpha subunit of the insulin receptor

8. Activation of insulin receptor tyrosine kinase

3. Phosphorylation of IRS proteins

6. Phosphorylation of phosphinositide 3-kinase (PI-3K)

4. Conversion of PIP2 to PIP3

7. Activation of PIP3-dependent protein kinase B (PDK1)

5. Glut4 receptors transported to the cell membrane

Explanation:

The insulin signaling pathway is described as under:

RTK (receptor tyrosine kinases) which is a receptor for insulin is an extracellular receptor but in contrast to other cell surface receptors it is catalytic in nature. In the absence of insulin (ligand), it is monomeric but as soon as it gets activated (activation occurs upon ligand binding), it undergo dimerization. It leads to auto-phosphorylation in it's tyrosine residue which subsequently leads to phosphorylation of tyrosine residue of other receptors. Such hyper-phosphorylated receptor have high affinity with enzyme/molecule like IRS protein which have SH2 domain . IRS down stream activates phosphinositide 3-kinase (PI-3K). This enzyme converts component of animal cell membrane PIP2 into PIP3. PIP3 also remains membrane bound but it has the potential to phosphorylate another enzyme named as PIP3-dependent protein kinase B (PDK1). Further, PDK1 leads to the activation of Akt or PK-B. Akt is a serine-threonine kinase which ultimately leads to the recruitment of Glut4 receptors on cell membrane for uptake of more and more glucose into the cell.

Note: Apart from this Akt also phosphorylates another protein named as FOXO which ultimately causes cell growth, Akt can also phosphorylate BAD protein so as to restrict cell apoptosis or we can say it leads to cell survival, Akt also leads to translation in a cell with the help of mTOR raptor etc.  

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3 years ago
Please answer quickly!
kirill [66]
By looking at the diagram, and my knowledge of Punnett squares, i would say B. 4:16
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Oie bom by eu também não sei
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2 years ago
How do nurse researchers strive for constancy of conditions in implementing a treatment?
zhenek [66]
<span>Having standard written research protocols is the correct answer. ;)</span>
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