<span>Glucose metabolism is disturbed by hypercortisolism and the client's fasting blood glucose is increased. The client also has decreased serum potassium level. Since cortisol is an immunosuppressant, it lowers the immune system by lowering the lymphocyte count. </span>
The answer would be C. Plantae because it has membrane bound organelles and green chloroplasts, as well as containing a nucleus.
Answer:
X-linked recessive inheritance
Explanation:
Since the disease is primarily present in males vs females, it is a s.ex linked disease.
In X-linked recessive inheritance, males tend to get disease b/c they only have one x (the other is y) which means if they get the bad "x" copy from mom then they definetely will get the disease.
Females can be carriers b/c they are XX. If they get a good copy from mom and suppose a bad copy from dad, they can still be healthy b/c the good copy is dominant. They will be carriers (cuz they have the bad copy) but themselves wont have the disease.
It is A, the cell is divided into a parent cell and daughter cell (mitosis)
Options for part A are as follows:
A) A mutation in the operator sequence
B) A mutation in the lac-Z gene
C) A mutation in the lac-Y gene
D) A super repressor mutation
Answer:
The correct answer:
Part a - A mutation in the operator sequence
Part b - It ensures that a cell dedicates resources to the production of enzymes involved in lactose metabolism only when lactose is available in the environment
Part C. true.
Explanation:
part a:
If there is a mutation in the operator sequence leads to prevent binding of the repressor which leads to allowing constitutive expression of the genes various conditions.
part b:
The biological role of the lac operon makes sure that the cell dedicates resources to the production of enzymes involved in lactose metabolism only when lactose is available in the environment
Part c:
RNA polymerase cannot transcribe the structural genes due to the repressor binds to the lac operator, therefore, the proper function of the lac operon is possible when the placement of the operator sequence between the promotor and the structural genes.
