Answer:
The number of offspring produced is often related to the amount of parental care. Typically, the higher fecundity, the lower the amount of time parents devote to caring for the offspring.
The answer is epididymis.
<span>Testicles contain seminiferous tubules, in which sperm is produced, and an epididymis, in which sperm matures. Mature sperm passes through the epididymis to the vas deferens. There, it combines with a liquid secretion from the seminal vesicles and continues through the prostate gland to the urethra. Sperm containing fluid is called semen and it is ejected through the urethra.</span>
Answer: Baboon, Leopard, Cheetah, Brown and Spotted Hyena, Klipspringer, Springbok, Steenbok, Cape and Bat Eared Fox, Hartmann's Zebra, as well as many insects, reptiles, small mammals and even wild Desert Horses
Explanation:
Answer:
The correct answer is e. All of the above.
Explanation:
Sister chromatids are made during the synthesis phase of the cell cycle. In the synthesis phase, the homologous chromosomes get replicated and sister chromatids are produced so they are produced by duplication of chromosomes.
As sister chromatids are produced by replication, therefore, they are identical copies of parent chromosomes. These sister chromatids are joined to each other at centromere. They get separated during the anaphase of mitosis and moves to the opposite pole.
Therefore the right answer is e.
Answer:
(A) It prevents electron flow from the iron-sulfur centers in complex 1 to the ubiquinone. Due to reduction in electron transfer rate, there is a decrease in the production of ATP which is dangerous for some insects and fish over time.
(B) It also prevents electron flow from cytochrome b to cytochrome c1 at the complex III which leads to QH2 accumulation. If oxidized Q is not present, these is alteration of electron flow and the production of ATP is altered.
(C) Rotenone only prevent electron transfer into the chain at Complex 1 but it does not affect electron transfer at Complex II. Although there is slow ETC, it does not stop completely. However, Antimycin A prevents the oxidation of QH2, the final electron acceptor crom complex I and complex II. Thereby, stopping the production of both ETC and ATP. It can be concluded that antimycin A is a more potent poison.
Explanation:
Rotenone prevents electron flow from the iron-sulfur centers in complex 1 to the ubiquinone. Due to a reduction in electron transfer rate, there is a decrease in the production of ATP which is dangerous for some insects and fish over time. Antimycin A also prevents electron flow from cytochrome b to cytochrome c1 at the complex III which leads to QH2 accumulation. If oxidized Q is not present, there is an alteration of electron flow and the production of ATP is altered. Antimycin A is more potent than rotenone.
