Hyperthymia or hyperthymic syndrome is a condition in which the individual possesses a superior autobiographical memory, such as to allow the memory of most of the events lived in one's existence.
Oceans often get oil and toxic waste dumped into them as a bit of a "cheat" to cheaply dispose of waste. This harms the ocean life greatly. A leading cause of dumping into the oceans is the expense of getting rid of used oil. If a city were to allow residents to dispose of these things at no cost to them, there would be no need for people to dump it into the ocean. This will reduce the harm to sea life.
Answer:
True
Explanation:
some genetics can cause mutations and can be passed down or skip generations
Answer:
A. Heritable
C. Affected by the MC1R gene
D. Polygenic trait
Has evolved due to evolutionary processes such as natural selection.
Explanation:
Human skin color is a polygenic trait as it is regulated by more than one gene. Each allele of these genes has an additive effect on the overall skin color of an individual. Therefore, the trait is a continuous trait, not a discrete one. The genes for human skin color are transmitted from parents to progeny which in turn makes it a heritable trait.
The gene MC1R codes for melanocortin 1 receptor and thereby, regulate the skin color of humans since melanocyte-stimulating hormone binds to the receptor to trigger the synthesis of melanin from melanocytes. Evolution of various skin color in human beings is regulated by natural selection which in turn favors the skin tone best suited under the prevailing climatic conditions of a particular human population
Answer:
Reduce or stop the production of energy molecule i.e ATP
Explanation:
Proton is an essential ingredient to carry out light reactions successfully and the proton requirement is fulfilled through the proton gradient formed along the membrane. A chemical that blocks the flow of electrons thereby hindering the flow of proton will adversely affect the electron transport chain reactions thereby causing reduced/no conversion of ADP to ATP.