Radioprotective agents may be useful as an adjunct to medical imaging to ... compared to those who underwent the same radiation exposure at an older age.
Answer:
The onset, and treatment of PTSD is no easy matter. In order for such a disorder to appear, which is later expressed with tremendous peaks of stress, crisis, behavioral problems, panic disorders, anxiety disorders, and many more debilitating conditions, a person must have experienced an event, or circumstance, that traumatically negatively impacted the correct psychological and emotional processes of the mind. In these patients, one the most common situations is that panic attacks and anxiety peaks happen particularly when the traumatic event, moment, or circumstance is remembered, and they are very good at remembering not just the moment itself, but all elements that surrounded that trauma. Also, they are able to make really clear connections and associations between those events and normal events in life that, when they appear, trigger the defensive responses of the mind.
The idea of administering a patient with PTSD with medication that would stop memory formation shortly after a traumatic event would not be a good idea simply because while the drugs focus on the chemical processes involved in memory formation, this procedure ignores that memory is much more than just chemicals in the brain. This has been show time and time again in research. Even with strong medication that basically generates amnesia, or trauma to the brain that may cause amnesia, it has been seen, and science cannot yet fully explain how, these patients may have partial, or total recovery of memories. And because the process of memory formation is still so misunderstood, and not all the factors involved are completely known, simply administering a drug that would stop the hormones that are believed to play a role in memory formation could become more of a problem for the patient. Also it is not being considered that amnesia causes even more stress in patients, as somehow the brain still recognizes that there is information present, but cannot simply bring it to concious awareness.
We do not see the world in black and white; neither do we see it as two-dimensional (2-D) or flat (just height and width, no depth). Let’s look at how color vision works and how we perceive three dimensions (height, width, and depth).
Color Vision
Normal-sighted individuals have three different types of cones that mediate color vision. Each of these cone types is maximally sensitive to a slightly different wavelength of light. According to the trichromatic theory of color vision, shown in Figure 1, all colors in the spectrum can be produced by combining red, green, and blue. The three types of cones are each receptive to one of the colors.
The trichromatic theory of color vision is not the only theory—another major theory of color vision is known as the opponent-process theory. According to this theory, color is coded in opponent pairs: black-white, yellow-blue, and green-red. The basic idea is that some cells of the visual system are excited by one of the opponent colors and inhibited by the other. So, a cell that was excited by wavelengths associated with green would be inhibited by wavelengths associated with red, and vice versa. One of the implications of opponent processing is that we do not experience greenish-reds or yellowish-blues as colors. Another implication is that this leads to the experience of negative afterimages. An afterimage describes the continuation of a visual sensation after removal of the stimulus. For example, when you stare briefly at the sun and then look away from it, you may still perceive a spot of light although the stimulus (the sun) has been removed. When color is involved in the stimulus, the color pairings identified in the opponent-process theory lead to a negative afterimage. You can test this concept using the flag in Figure 2.
But these two theories—the trichromatic theory of color vision and the opponent-process theory—are not mutually exclusive. Research has shown that they just apply to different levels of the nervous system. For visual processing on the retina, trichromatic theory applies: the cones are responsive to three different wavelengths that represent red, blue, and green. But once the signal moves past the retina on its way to the brain, the cells respond in a way consistent with opponent-process theory (Land, 1959; Kaiser, 1997).
Depth Perception
Our ability to perceive spatial relationships in three-dimensional (3-D) space is known as depth perception. With depth perception, we can describe things as being in front, behind, above, below, or to the side of other things.
Our world is three-dimensional, so it makes sense that our mental representation of the world has three-dimensional properties. We use a variety of cues in a visual scene to establish our sense of depth. Some of these are binocular cues, which means that they rely on the use of both eyes. One example of a binocular depth cue is binocular disparity, the slightly different view of the world that each of our eyes receives.
A 3-D movie works on the same principle: the special glasses you wear allow the two slightly different images projected onto the screen to be seen separately by your left and your right eye.
Although we rely on binocular cues to experience depth in our 3-D world, we can also perceive depth in 2-D arrays. Think about all the paintings and photographs you have seen. Generally, you pick up on depth in these images even though the visual stimulus is 2-D. When we do this, we are relying on a number of monocular cues, or cues that require only one eye. If you think you can’t see depth with one eye, note that you don’t bump into things when using only one eye while walking—and, in fact, we have more monocular cues than binocular cues.
An example of a monocular cue would be what is known as linear perspective. Linear perspective refers to the fact that we perceive depth when we see two parallel lines that seem to converge in an image (Figure 3).
Vision is not an encapsulated system. It interacts with and depends on other sensory modalities. For example, when you move your head in one direction, your eyes reflexively move in the opposite direction to compensate, allowing you to maintain your gaze on the object that you are looking at. This reflex is called the vestibulo-ocular reflex. It is achieved by integrating information from both the visual and the vestibular system (which knows about body motion and position). You can experience this compensation quite simply.
Finally, vision is also often implicated in a blending-of-sensations phenomenon known as synesthesia.
SORRY ITS A LONG ANSWER!!!
A client is diagnosed with an st segment elevation myocardial infarction (stemi) and is receiving a tissue plasminogen activator, alteplase. monitoring for signs of bleeding is the priority nursing intervention.
<h3>Tissue Plasminogen activator:</h3>
It is a thrombolytic. symptomatic and systemic hemorrhage is a complication of any type of thrombolytic medication.
Tissue Plasminogen activator is an enzyme which is useful to treat diseases which includes heart attack, strokes and blood clots ,etc.
It is one type of glyco-protien which produced mainly a vascular endothelial cells.
The side effects of tissue Plasminogen activator includes headache, bleeding from the wound, paralysis, difficulties in breathing and swallowing, etc.
Alteplase is a fibrinolytic agent . it is also referred to as tissue plasminogen activator (tPA). Alteplase converts plasminogen to the proteolytic enzyme plasmin, which lyses fibrin as well as fibrinogen.
<h3>What is blood clots? </h3>
Blood clots are gel-like thick collections of blood that form in your veins or arteries when blood changes from liquid to partially solid.
A blood clot is also called a thrombus.
Learn more about blood clots here:
brainly.com/question/8204597
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