Answer:
6 thymine, 4 guanine. 4 cytosine
Explanation:
Adenine pairs with thymine, so you need 6 of each
Cytosine matches with guanine, but the total of nucleotides is 20, so
6(adenine)+6(thymine)=12
20-12=8
8= 4(guanine)+4(cytosine)
On the off chance that I was a safeguard lawyer contending in court for shielding a coral reef from hurtful human exercises I would give particular contentions in the guard of this environment in the matter of why it must be ensured. I would first delineate that despite the fact that the coral reefs possess just 0.2% of the sea floor they give such imperative biological administrations. They go about as a characteristic boundary and ensure around 15% of the world's coastlines that would be harmed and devastated by disintegration from waves and tempests. They additionally give living spaces to about a fourth of the majority of the marine living beings. Next, I would depict how financially imperative coral reefs are. Coral reefs make up about a tenth of the world's worldwide fish catch and one-fourth of fish found inside creating nations.
Numerous degenerative neurological conditions, most notably Parkinson's disease, have been linked to an excessive buildup of alpha synuclein (a-syn) in the brain. Intraneuronal inclusions, often known as Lewy bodies, are neuropathological characteristics seen in Parkinson's disease, Lewy body dementia, and other synucleopathies. The aggregation of a-syn is their main structural component. A-syn accumulation, aggregation, and ensuing Lewy body formation can be attributed to a variety of biological processes. These include genetic changes in parkin, synuclein, or the deubiquitinating enzyme ubiquitin C-terminal hydrolase (UCH-L1), which results in less efficient removal of a-syn via the ubiquitin proteasomal pathway (UPP). Additionally, environmental variables and an age-related decline in antioxidant defense mechanisms that heighten oxidative stress and can have an impact on the formation or clearance of a-syn are intracellular insults.
We focused on changes in the aggregation and clearance of a-syn as impacted by the UPP and the oxidative stress pathways in our dynamic models of a-syn processing in both normal and various disease states. A free radical profile similar to that observed in vivo after exposure to the neurotoxin 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine is produced during simulation of enhanced oxidative stress (MPTP). To replicate the kinetics of a-syn that correlates to the neuropathology reported for the sporadic and hereditary types of Parkinson's disease, different model parameters of oxidative stress, UPP failure, or both routes are used. With the use of this in silico model, it is possible to evaluate the kinetics of pathway elements and more accurately identify and validate key pharmaceutical targets.
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Answer:
DNA replication is the process by which a molecule of DNA is duplicated. When a cell divides, it must first duplicate its genome so that each daughter cell winds up with a complete set of chromosomes.
Explanation:
