They create competition with some certain niche
Algae because it is a small organism that the larva state of animals live in and feed on it
The answer is vitamin D. This is actually a group of vitamins of similar molecular structure responsible for facilitating increased absorption of calcium, magnesium and phosphate from the gut. The most important of the vitamin D compounds are vitamin D3 and D2. While vitamin D can be obtained from your diet and supplements, the major source of vitamin D is from a chemical reaction dependent on skin exposure to the sun.
Okazaki fragments are the discontinuous segments of the lagging strand.
Okazaki fragments are located on the template strand which dictates the newly synthesized DNA away from the direction of the movement of replication fork. It is the building block for DNA synthesis of the lagging strand and on one template strand, the DNA polymerase synthesizes the new DNA in the opposite direction that is away from the replication fork movement.
Answer:
The colonies are carrying the resistance genes from plasmids
Explanation:
Bacteria can acquire beneficial characteristics that they didn’t have. One way for these is through plasmids, which ones are little fragments of DNA that usually contains resistance genes (for antibiotics, disinfectants, heavy metals, etc.) or other capacities, like the ability to use some substances (for example sugars).
In this specific situation, we already know that the plasmid carrying genes for tetracycline resistance and the <em>lacZ</em> gene.
A little explanation:
Tetracycline is an antibiotic that inhibits bacterial growth and kills the bacteria. The bacteria can “fight” to this antibiotic if it has a resistant gene, the result is that the antibiotic can’t affect the bacteria and survive. An analogy is like a Police Officer (bacteria) that have a bulletproof vest (tetracycline-resistant gene) so the bullets (tetracycline) didn’t affect the police.
In the case of X-gal, is a compound consisting primarily in one sugar called galactose. Not all bacteria can eat galactose, they need an enzyme called β- galactosidase (comes from <em>lacZ</em> gene) that helps the bacteria “eat” the sugar (cuts the sugar in little pieces so the bacteria can eat).
Then, as the bacterial colonies can grow in the medium with tetracycline and X-gal, we know that those bacteria are carrying the resistance genes for tetracycline (does not affect the bacteria) and the <em>lacZ</em> gene (bacteria produce β- galactosidase that cuts galactose). These genes are coming from the plasmids because we already know that the plasmid carries these genes and not from the exogenous DNA.
