Answer:
The correct answer is C production of FAD to FADH2 by the citric acid cycle represent the energetic capacity to synthesize about 1.5 ATP.
Explanation:
Citric acid cycle is one of most important stage of cellular respiration.Citric acid cycle or TCA cycle composed of various enzyme catalyzed biochemical reaction.
various reduced coenzymes are produced during TCA cycle such as NADH,FADH2.FADH2 is formed from FAD during the conversion of succinate to fumarate.
The FADH2 then enter electron transport chain to oxidize itself into FAD along with the generation of 1.5 ATP.
When an individual is dehydrated, his or her intravenous fluids should be isotonic, because either a hypertonic or hypotonic IV would both cause damage to the individual’s red blood cells. Isotonic solutions are used: to increase the EXTRACELLULAR fluid volume because of blood loss, surgery, dehydration, fluid loss that has been loss extracellularly.
The original roots of dodder usually die. As a parasitic plant that cannot produce its own chlorophyll, it breaks away from its root system as the roots has no apparent root cap nor apical meristems. Hence the "root" is not performing its very function of water and nutrient uptake. It then produces haustoria, a root-shaped fungi which penetrates host plant which makes the dodder parasitic.
They return nutrients from waste or dead organisms back into the ecosystem to be recycled.
Answer:
The old idea that coronary heart disease is an infectious disease has gained popularity in recent years, and both viral and bacterial pathogens have been proposed to be associated with the inflammatory changes seen in atherosclerosis. Herpes group viruses, notably cytomegalovirus and herpes simplex type 1, have been associated with atherosclerosis and restenosis. Helicobacter pylori and dental infections have also been linked to atherogenesis, but the evidence seems to favor a respiratory, obligatory intracellular bacterium, Chlamydia pneumoniae. The association was originally found in seroepidemiological studies, but the actual presence of the pathogen in atherosclerotic lesions has been repeatedly demonstrated, and during past year the first successful animal experiments and encouraging preliminary intervention studies were published. The causal relationship has not yet been proven, but ongoing large intervention trials and continuing research on pathogenetic mechanisms may lead to the use of antimicrobial agents in the treatment of coronary heart disease in the future.
Explanation:
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