Both high levels and very low levels of insulin lead to increased eating because little glucose is reaching the cells to be used as fuel.
<h3>How do insulin levels affect apatite?</h3>
Cells will absorb too much glucose from the blood if a person's insulin level rises over what is necessary, such as when they spend more energy than expected or eat less than they had planned. An abnormally low blood glucose level (hypoglycemia) results from this . As a response to hypoglycemia, the body releases liver-stored glucose in an effort to restore normal blood sugar levels. Elevated insulin levels result in greater appetite, enhanced enjoyment of sweet tastes, and higher food intake.
Type 1 diabetics either produce very little insulin or none at all. This illness is brought on by the beta cells' inability to produce insulin due to antibody destruction (antibodies are typically molecules the body releases to fight infections). High blood glucose levels result from the body's inability to transport glucose from the blood into the cells when there is insufficient insulin in the body. If the glucose is not reaching the cells, the person will eat more.
I understand the question you are looking for is this:
Why do both high levels and very low levels of insulin lead to increased eating?
- Glucose leaves the blood to be stored as fat.
- Fat supplies are being rapidly converted to glucose.
- Activity of the taste buds is directly enhanced.
- Little glucose is reaching the cells to be used as fuel.
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