Answer:
polar regions and nonpolar regions
Explanation:
if right braniliest? I only need one more
Answer:
Prophase 1
Explanation:
Prophase I :It can be divided into five stages:
1)Leplotene: In this stage, chromosome becomes visible and shorten as a single structure.
2)Zygotene: Homologous / sister chromosome pair up. This process is known as synapse. Synapse is the process of attachment of homologous /sister chromosome. One pair comes from the male parent while the other comes from the female parent.
3)Pachytene: Bivalents shorten.
4)Diplotene:Point of attachment of non-sister chromatids. The two chromosomes are seen to be joined at several points along their length. These points are known as Chiasmata.(Chiasmata, a cross). The paternal gene A, B, C may swap for the maternal gene a, b, c for instance, leading to a new gene combination in the resulting chromatids. This is referred to as *CROSSING OVER*.
5)Diakenesis: Homologous centromere move apart.
So, crossing over occurs in *Prophase 1.
A researcher is interested in screening for p-elements inserted into a region on chromosome 3. to produce new insertions, she crosses females homozygous for a miniwhite + p-element on the x chromosome with males homozygous for immobilized transposase on chromosome 2. what genotype will the f1 male flies have? (note that a "+" indicates a wild-type chromosome without the transposase locus or the p {miniwhite +} insertion. the x /y chromosomes are listed first, followed by the second
Answer:
Cytokines made by other innate cells, such as macrophages or dendritic cells
Explanation:
Innate Pattern Recognition Receptors (PRRs) on Dendritic cells and macrophages sense and respond to PAMPs and produce cytokines. Cytokines start the innate immune response.
The cytokines are produced in response to pathogen-associated molecular patterns (PAMPs) such as peptidoglycan monomers or CpG sequences on bacterial and viral genomes. Cytokines produced in response to pathogen receptors on cell surfaces, such as the inflammatory cytokines IL-1, IL-6, IL-8, and TNF-alpha, chemokines and type I interferons that act on leukocytes so as to promote and control early inflammatory responses.
