Answer:
I have no clue what that means sorry wish I could help
Answer:
Answer explained
Explanation:
a. Overexpression of Axl receptor tyroxine kinase correlates with the decresed patient's overall survival and hence it's inhibition is under process.
AXL TKI BMS777607 treatment increased AXL protein levels after 24 h as observed by Western blot and flow cytometry analysis. Mechanistically, this inhibition-induced AXL cell surface accumulation was neither associated with epigenetic modifications, nor altered transcriptional and translational regulation. Hence this later increased the glycosylated protien abundance in kinase dead mutant. The kinase function blockaging resulted in impaired internalisation which finally incresed the level of proliferation.
An alternative technique may exist in linkage of Axl Tkl to a degradation machinery recruitment unit .
b. Mendelian disorders like cystic fibrosis results from error in the given DNA sequence which can be autosomal dominant and recessive both and also X or Y linked. Mutated gene results in the Mendelian disorders. Whereas in cancer there can be mutation or proliferation of the given cells in a specific area. In cancer cells there is 2.5 times more the mRNA present while 6.2times the DNA and hence the detection and diagnosis automatically becomes difficult as in Mendelian disorders only single sequence needs to be studied at a time .
Answer:
<em>Option "A" might be the correct answer</em>
<em>Explanation:</em>
<em>there are some of those viruses where it can be quite potent. Some viruses, such as the rhinoviruses, seldom cause serious illness. But others, such as parainfluenza and respiratory syncytial virus, can lead to severe lung infections in young children.</em>
As covalent bonds are strong it makes the sugar phosphate backbone a stable molecule which will HV less chances for a mutation while the huydrogen bonds exist for the ease of DNA replication as they are strong in a way but weak for the replication to take place easily.
Answer:
biosphere is the answer bro
