Answer:
The possible fate of the cell that it may turn cancerous.
Explanation:
The cells present in the body generally work in harmony. However, if a cell attains a mutation, it can make it proliferate in the case when it should not do, and make it thrive in the case when other cells are dying. Due to proliferation, the unusual cell produces more abnormal cells also known as cancerous cells. These cancerous cells become more favorable in comparison to the normal cells due to the phenomenon of natural selection. These cells eventually result in a lethal form of tumors.
In the normal cells, the destructed gene or the damaged cells get repaired easily, in case if the damage is worse the cell dies. A protein known as p53 helps in repairing damaged cells or kills them if the damage is too severe. But in the case of cancer cells, the p53 protein does not work appropriately as they possess a mutated or changed form of p53 protein. Thus, in the case of cancerous cells, the rate of repair lags behind the rate of mutation, which makes the cancer cells thrive and increase in numbers resulting in further destruction.
Neurotransmitter has to be consumed in the diet is not the criteria for neurotransmitter. So the correct option is a.
A neuron releases a signaling chemical called a neurotransmitter across a synaptic gap to influence another cell. Any major body component or target cell that receives the signal may be another neuron, but it could also be a gland or muscle cell. These signals enable you to move your limbs, experience sensations, maintain heartbeat, as well as receive and process all the information your body receives from other internal body parts and your environment.
These signals enable you to move your limbs, experience sensations, maintain heartbeat, as well as receive and process all the information your body receives from other internal body parts and your environment.
The axon terminal, a component of the neuron, is where neurotransmitters are found. They are kept inside synaptic vesicles, which have thin walls. Numerous thousands of neurotransmitter molecules can fit inside each vesicle.
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If yellow seed color is primarily dominant, future generations will likely turn out yellow as well due to the relationship between dominant and recessive genes. Dominant genes and traits are mostly more likely to show in future generations.
<h2>Mitotic Cell Cycle</h2>
Explanation:
a. Cytochalasin: an inhibitor of actin microfilament
- Cytokinesis is the process by which cytoplasm is divided into two cells hence generation of two daughter cells
- In animal cells the division of cytoplasm starts with the assembly of contractile ring
- Contractile band consists of actin and myosin and catalyze cleavage furrow formation
- Cytochalasin is a drug that blocks the polymerization of actin filament
- When cytochalasin is added dividing cell fails to undergo in cytokinesis due to defective assembly of contractile band
b. Colchicine: an inhibitor of microtubule formation
- Colchicine tightly binds with free tubulin dimer and prevents its polymerization
- In this case mitotic spindle apparatus do not assemble and cells unable to do partitioning of chromosomes into two groups
- Thus cell division is arrested
c. Aphidicolin: an inhibitor of DNA Polymerase activity
- Aphidicolin is used to induce cell cycle arrest via specific inhibition of DNA Polymerase α
- It blocks the cell cycle at early synthesis(S) phase
d. Emetine: an inhibitor of ribosome activity
- It blocks the protein synthesis in eukaryotic cells by binding to small subunit(40 S) of ribosomes
- It interferes with the synthesis and activities of DNA and RNA
<span>Gene expression would be the answer.</span>
