Answer:
How do policy and management decisions affect the levels of the primary carbon-containing gases, carbon dioxide and methane, in the atmosphere?
nobody realy know but we so know that it is alwas getting bigger
Answer:
b. Only about one-half of the world's population relies on sunlight to maintain adequate vitamin D nutrition
Explanation:
Dark skin pigments slow down the synthesis of vitamin D, so black people need to spend more time in the sun to absorb enough vitamin D, so the letter A is incorrect.
Prolonged exposure to sunlight degrades the precursor of vitamin D, whereas prolonged exposure to sunlight produces vitamin D3, which acts to regulate heart function and blood pressure. With this we can conclude that the letter C is also incorrect.
Vitamin D deficiency is not caused by calcium deficiency, on the contrary, vitamin D deficiency is a cause of calcium deficiency. In addition, summer vitamin D synthesis stocks are usually not sufficient to meet summer vitamin D requirements. That is why the letter D is also incorrect.
We can conclude, then, that the correct answer is the letter B. The main source of vitamin production is through sun exposure, because type B ultraviolet rays (UVB) are able to activate the synthesis of this substance. Some foods, especially fatty fish, are sources of vitamin D, but the sun is responsible for 80 to 90% of the vitamin the body receives. It can also be produced in the laboratory and given as a supplement when there is a deficiency and for the prevention and treatment of a variety of diseases.
Answer:
This disorder is caused by mutations in either of two different genes; one of these genes is linked to the DNA marker locus and the other gene is not.
Explanation:
A gene marker (also known as a molecular marker) is defined as a particular DNA sequence that has a specific physical location on a given chromosome. The gene markers are always physically linked with genes of interest, thereby they are useful for identifying patients affected by a given disease and/or a particular phenotypic trait. In this case, in the first study, genetic variation of the marker has been shown to be useful to map one <em>locus</em> that is associated with the disease phenotype. However, in the second study, a second <em>locus</em> that is not linked to the marker can segregate independently, thereby showing different results from the first study (in this case, it is imperative to develop a new genetic linkage analysis to discover genetic markers linked to the second gene, which is also responsible for the disease phenotype).
