The thorax is a very vital and important organ in the human body.
- It is a structure that serves as a<u> </u><u>protective covering</u> for organs in the body such as<u> the lungs, the heart, some vital blood vessels</u> e.t.c
It is<u> false</u> that to <em>make a person breathe in, the volume of the chest cavity must decrease, creating a higher pressure in the thorax.</em>
- When a person breathes in, <u>the chest cavity expands (the volume increases) and the pressure in the thorax decreases.</u>
- When a person breathes out, <u>the chest cavity decreases in size, and the pressure in the thorax increases. </u>
- Therefore, we can see that the <u>process of breathing in humans</u>, follows the <u>principle of Boyle's Law.</u>
- Boyle's law states that as the<u> volume increases there is a decrease in pressure</u> and <u>as the volume decrease there is an increase in pressure. </u>
Therefore, It is <u>false</u> that to <em>make a person breathe in, the volume of the chest cavity must decrease, creating a higher pressure in the thorax.</em>
To learn more, visit the link below:
brainly.com/question/24359876
Osmosis<span> is the phenomenon of the movement of Solvent molecules from lower solute conc. to higher solute conc. through a semi-permeable membrane to make solute conc. equal on both sides.</span>
Answer:
PFFT this might help? sorry if not mate
Explanation:
Cell cycle checkpoint controls play a major role in preventing the development of cancer [see Sherr, 1994, for a more detailed discussion]. Major checkpoints occur at the G1 to S phase transition and at the G2 to M phase transitions. Cancer is a genetic disease that arises from defects in growth-promoting oncogenes and growth-suppressing tumor suppressor genes. The p53 tumor suppressor protein plays a role in both the G1/S phase and G2/M phase checkpoints. The mechanism for this activity at the G1/S phase checkpoint is well understood, but its mechanism of action at the G2/M phase checkpoint remains to be elucidated. The p53 protein is thought to prevent chromosomal replication specifically during the cell cycle if DNA damage is present. In addition, p53 can induce a type of programmed cell death, or apoptosis, under certain circumstances. The general goal of p53 appears to be the prevention of cell propagation if mutations are present. The p53 protein acts as a transcription factor by binding to certain specific genes and regulating their expression. One of these, WAF1 or Cip1, is activated by p53 and is an essential downstream mediator of p53-dependent G1/S phase checkpoint control. The function of p53 can be suppressed by another gene, MDM2, which is overexpressed in certain tumorigenic mouse cells and binds to p53 protein, thus inhibiting its transcriptional activation function. Other cellular proteins have been found to bind to p53, but the significance of the associations is not completely understood in all cases. The large number of human cancers in which the p53 gene is altered makes this gene a good candidate for cancer screening approaches.
False because deciduous forests are located primarily in the eastern half of the United States,Canada<span>, Europe, parts of </span>Russia<span>, </span>China<span>, and </span>Japan<span>.</span>