Answer:
Outside
Explanation:
Newton's First Law of Motion states that a body at rest will remain at rest unless an outside force acts on it, and a body in motion at a constant velocity will remain in motion in a straight line unless acted upon by an outside force. ... Outside forces are sometimes called net forces or unbalanced forces.
If you change more than one variable at a time, then you will have a harder time trying to figure out why the certain change occurred.
Long-term potentiation (LTP) is considered a cellular correlate of learning and memory. The presence of G protein-activated inwardly rectifying K(+) (GIRK) channels near excitatory synapses on dendritic spines suggests their possible involvement in synaptic plasticity. However, whether activity-dependent regulation of channels affects excitatory synaptic plasticity is unknown. In a companion article we have reported activity-dependent regulation of GIRK channel density in cultured hippocampal neurons that requires activity oF receptors (NMDAR) and protein phosphatase-1 (PP1) and takes place within 15 min. In this study, we performed whole-cell recordings of cultured hippocampal neurons and found that NMDAR activation increases basal GIRK current and GIRK channel activation mediated by adenosine A(1) receptors, but not GABA(B) receptors. Given the similar involvement of NMDARs, adenosine receptors, and PP1 in depotentiation of LTP caused by low-frequency stimulation that immediately follows LTP-inducing high-frequency stimulation, we wondered whether NMDAR-induced increase in GIRK channel surface density and current may contribute to the molecular mechanisms underlying this specific depotentiation. Remarkably, GIRK2 null mutation or GIRK channel blockade abolishes depotentiation of LTP, demonstrating that GIRK channels are critical for depotentiation, one form of excitatory synaptic plasticity.
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Answer:
pulmonary vein
Explanation:
The pulmonary veins
transfer oxygenated blood from the lungs to the heart.
It helps complete the pulmonary circuit, by receiving oxygenated blood in the alveoli and returning it to the left atrium.
Answer:
Viruses are like hijackers. They invade living, normal cells and use those cells to multiply and produce other viruses like themselves. This can kill, damage, or change the cells and make you sick. Different viruses attack certain cells in your body such as your liver, respiratory system, or blood.
Explanation:
Viruses tend to target specific tissues (cells) in the host.
For example, the influenza virus has a predilection for the respiratory tract, hepatitis viruses target the liver, polio virus targets the motor neurons of the spinal cord and rotavirus multiplies in the gut. Symptoms of a viral infection may be subtle and nonspecific or specific and suggestive of the causative agent.
Dengue virus, Ross river virus, measles and rubella infections are associated with fever and a widespread red rash, chicken pox and herpes simplex viruses are associated with blistering, often localized, rashes; and hepatitis viruses cause liver damage and jaundice.
Bacteria tend to be less tissue-specific and non-discriminatory than viruses and can cause a variety of infections once they have invaded the host.
These bacterial infections are often manifested by the presence of pus wherever the bacteria settle, and systemic symptoms such as fevers, chills, pain, swelling and loss of function occur when bacteria invade and multiply.
