49.09 mL/hr, see attached photo for steps
Type because you are adding a different type of cardiovascular workout to your workout
Inhibiting cyclooxygenase-1 (COX-1) enzymes.
NSAIDs are one of the classes of medications that doctors most frequently prescribe. When using NSAIDs, patients frequently have stomach pain, and some of them go on to encounter major gastrointestinal issues such ulceration, bleeding, perforation, or blockage.
The majority of NSAID digestive adverse effects are related to COX inhibition, which lowers prostaglandin levels in the stomach mucosa. Arachidonic acid is transformed into prostaglandin (PG) H2, which is a precursor of PGs and thromboxane, by the enzymes cyclooxygenase-1 and cyclooxygenase-2 (COX-1 and COX-2).
These lipid mediators are crucial for both normal physiological processes and inflammatory and painful processes.
Here is another question with an answer similar to this about (COX-1) enzymes: brainly.com/question/19168320
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Answer: During a steady state each ion's driving force is O mv
Explanation:
When a cell is considered to be in steady state it means that the concentration ions and molecules within the cell is constant. As a result, a system at equilibrium is also considered to be at a steady state.
When in steady state, the membrane potential experiences no net change which is why the resting potential is an example of a steady state.
However during a steady state, each ion's driving force is not 0 mv.
