Answer: inability to regulate blood glucose levels.
Explanation:
None of the above. Neurotransmitters are chemicals like serotonin, norepinephrine and dopamine inside the brain and thus deal with the specialties of medicine, neurology and psychiatry. Hope that helps!
Answer:
The correct answer is option D. give furosemide.
Explanation:
Digoxin is a drug that uses in the cardiovascular condition. It inhibits the Na-K ATPase enzyme, which functions to maintain the intracellular environment by regulating the entry and exit of sodium and potassium.
Digoxin toxicity may occur due to the low level of potassium (inside the cell)due to the inhibitaion of the sodium-potassium pump. Furosemide is a diuretic drug that decreases salt concentration (such as ions) in the body by increasing urine output. and causes a low level of potassium in the body
Thus, furosemide increases digoxin toxicity as during low levels of potassium, digoxin can easily bind with the sodium-potassium pump and shows severe effects.
Answer:The final step in both pathways would be the activation of factor X, leading to conversion of prothrombin II to thrombin IIa. Option C.
Explanation: blood coagulation is the processes that leads to blood clot formation. The blood clot formed is made up of fibrin and platelets, which forms hemostatic plug over a wound site. The blood coagulation is made up of two partways that led to the formation of clot. The intrinsic pathway also called contact activation pathway and the extrinsic pathway which is also known as the tissue factor pathway. The both pathways are connected by a common pathway whereby factor X is activated leading to leading to conversion of prothrombin II to thrombin IIa. Thrombin then converts fibrinogen to fibrin( blood clot). I hope this helps. Thanks.
