Answer:
Statement A - cellular membrane
Statement B - eukaryote
Statement C - spore
Statement D - prokaryote
Explanation:
The cell membrane is a protective layer present around the cell and cell organelle that regulates and allows different molecules to enter and exit to regulate homeostasis.
Eukaryotes are the organism that can be unicellular or multicellular with the cells contain a nucleus in it.
Spores are reproductive cells that has a resistant layer that protects the cell from an unfavorable condition and capable to form an adult.
An organism that is unicellular and lacks the nucleus is known as a prokaryote and bacteria is an example of such organisms.
One of the species dieing off.
Answer:
In the traditional phylogeny platyhelminthes is depicted as a lophotrochozoan phylum. What probably led to this change?
The removal of the acoel flatworms (Acoela) from the Platyhelminthes allowed the remaining flatworms to be clearly tied to the Lophotrochozoa.
Explanation:
Answer:
Taxol induces the assembly of microtubules, while Nocodazole suppresses tubulin assembly
Explanation:
Taxol and nocodazole are antimitotic drugs, i.e., drugs that inhibit the progression through the cell cycle, which is useful for killing cancer cells. Microtubules are proteins that act as the 'skeleton of the cell', which need to be reorganized when cells replicate. These structures (microtubules) are composed of α and β-tubulin heterodimers which assemble into protofilaments of microtubules. Taxol is an antimitotic drug that induces the formation of microtubules, thereby inhibiting microtubules' reorganization during mitosis. In consequence, cells treated with Taxol enter into apoptosis (i.e., programmed cell death). Moreover, Nocodazole is known to induce microtubule disassembly by interfering with the polymerization of tubulin monomers. In consequence, Nocodazole is useful to depolymerize the microtubule cytoskeleton.
Answer:
Methotrexate inhibits the synthesis of DNA, RNA, and protein synthesis of the cells in the experiment.
Explanation:
Methotrexate competitively inhibits the enzyme dihydrofolate reductase (DHFR) which is involved in the synthesis of tetrahydrofolate from the inactive dihydrofolate. The methotrexate blocks the enzyme by binding to its active site thereby preventing further reaction of the dihydrofolate reductase enzyme and dihydrofolate. Tetrahydrofolate is a useful material for the de novo synthesis of thymidine, a nucleoside. And without thymidine, DNA synthesis is hindered. Methotrexate therefore inhibits the synthesis of DNA, RNA, thymidylates and of course protein synthesis.