Active transport is the process in which substances are absorbed against a concentration gradient, i.e. from a lower to a higher concentration.
Answer: Both the mouse and human beings are encoding the proteins having similar functions.
Explanation:
The CFTR gene in the mouse and CFTR gene in the human beings encode proteins that perform similar function,this is so because if the results are same then it means that both mouse and human beings are encoding the protein that have similar function.
Option A is incorrect because both humans and mouse are eukaryotes so they have introns and because of that even if the gene sequences are different they can still encode protein.
Option B is incorrect because amino acid sequences can be similar inorder to have the similar function. If glutamic acid is replaced by aspartic acid, it won't have different function the function will remain same. So, identical amino acid sequences are not required. They can be similar too.
False
reason -
Gene testing is not full proof as there are possibilities of discrepancies in it based on the testing procedures used and the condition of sample. Since we human beings have the same genomic structure there are chances that our DNA may match with other people who do not share a heredity with our family. Basically statistical probability is used to determine probability of paternity, relationship of any kind etc. among two individuals . usually A 99% or higher percentage of probability is considered conclusive. and thus it gene testing can not be considered as full proof
Explanation:
Almost all eukaryotic proteins are subject to post-translational modifications during mitosis and cell cycle, and in particular, reversible phosphorylation being a key event. The recent use of high-throughput experimental analyses has revealed that more than 70% of all eukaryotic proteins are regulated by phosphorylation; however, the mechanism of dephosphorylation, counteracting phosphorylation, is relatively unknown. Recent discoveries have shown that many of the protein phosphatases are involved in the temporal and spatial control of mitotic events, such as mitotic entry, mitotic spindle assembly, chromosome architecture changes and cohesion, and mitotic exit. This implies that certain phosphatases are tightly regulated for timely dephosphorylation of key mitotic phosphoproteins and are essential for the control of various mitotic processes. This review describes the physiological and pathological roles of mitotic phosphatases, as well as the versatile role of various protein phosphatases in several mitotic events.
