Answer:
The correct answer is given below:
Explanation:
Caenorhabditis elegans is a round worm. It is a microscopic organism that lives in the soil. It is regarded as a model organisms because its organ systems such as nervous, muscular and reproductive are similar as in higher animals such as mice and humans. 131 cells formed in the developing embryo die by the process of apoptosis. It is a programmed cell death. The cell death is not random; but which cells will die and at what stage is completely programed.
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How do I solve this?A large population of laboratory animals has been allowed to breed randomly for a number of generations. After several generations, 49% of the animals display a recessive trait (bb), the same percentage as at the beginning of the breeding program. The rest of the animals show the dominant phenotype, with heterozygotes indistinguishable from the homozygous dominants.
What is the frequency of allele b in the gene pool?
Answer: <span>0 .70</span>
Answer:
How do amino acid sequences provide information about evolutionary relationships among organisms? They allow scientists to compare the order of the acids in a protein to see how closely related the organisms are.
Explanation:
Small intestine
Peyer's patch are small masses of lymphatic tissue found usually in the ileum region of the small intestine. This is an aggregated lymphoid nodules and form an important function in immune system. This allows to monitor bacterial population and prevents the growth of pathogenic flora in the intestine. They are egg shaped lymphatic tissue that are similar to lymph nodes in structure except the absence of connective tissue.
Answer:
on a tyrosine residue
bind to insulin
Explanation:
The insulin receptor substrate-1 (IRS1) is a signaling protein that can be phosphorylated on multiple tyrosine and serine/threonine residues. IRS1 contains several conserved domains including a pleckstrin (PH) domain and a PTB domain involved in protein phosphorylation and ligand binding. In the first place, IRS1 is phosphorylated on a tyrosine residue, and then IRS1 binds to insulin or the insulin-like growth factor-1 (IGF-1), thereby activating transduction pathways such as, for example, MAPK/ERK. Moreover, RS1 is also phosphorylated on serine residues, thereby triggering opposite effects in insulin-associated signaling.
