Answer:
to prevent the virus to have a big effect on the body of humans so the cells can fight against it
Answer:
A. A majority of scientists agreed with it
Explanation:
The famous theory called the theory of common descent, states that all the living organisms of the earth have arisen from a common ancestor. This notion was first proposed by a French mathematician,Louis Maupertuis duirng 1740s who was of the view that all organisms had a single ancestor and evolutionary process with the passage of time resulted in the specie diversification.
After that, in 1790s another philosopher Immanuel Kant, suggested that all organisms seem to have a common ancestor. In the same period of 1790s, another scientist , Erasmus Darwin who was the grandfather of Charles Darwin also suggested that all the organisms might have a single ancestor who went through the process of evolution to bring all the majesty into life.
Charles Darwin was the first scientist who worked on this notion for alot of time and proposed the theory of common descent,in his book, On the Origin of Species.
After it, many scientists got agree with this theory such as, Vernon Kellogg in 1907 and T. Ryan Gregory in 2008 and many others explain that no reliable observations exists which contradicts the theory of common descent.
Therefore, option A is the best option.
Hope it helps!
Answer: Aldolase
Explanation:
In the metabolism of glucose( glycolysis) phosphofructokinase is an enzyme that catalyzes the conversation of fructose-6-phosphate into fructose-1,6-bisphosphate. This in turn is converted to pyruvate after various steps of enzymatic activity in the glycolytic pathway.
If phosphofructokinase experienced a mutation that interfered with substrate binding, the enzyme that is going to be most immediately impacted in terms of accessing substrate is the ALDOLASE.
Aldolase enzymes cleave fructose-1,6-bisphosphate to triose phosphates( glyceraldehyde 3-phosphate and dihydroxy-acetone phosphate) facilitating an increase in anaerobic production of ATP in muscle.
Therefore, the substrate for binding of aldolase, which is fructose-1,6-bisphosphate is lacking due to mutation of phosphofructokinase enzyme.
