A low serum magnesium level (less than 1.46 mg/dL) in the blood results in hypomagnesemia, an electrolyte disorder. Hypomagnesemia may result in serious consequences that result in abrupt death, ventricular arrhythmia, and coronary artery spasm.
Chronic illness, alcohol use disorder, gastrointestinal losses, renal losses, and other disorders can all contribute to hypomagnesemia.
<u>Warning signs of </u><u>hypomagnesemia</u>
- Tremors.
- Tetany is characterised by muscle cramps, spasms, and/or numbness in the hands and feet.
- inconsistent eye motions (nystagmus).
- weakness and exhaustion.
When a magnesium deficit is symptomatic or chronically below 1.25 mg/dL ( 0.50 mmol/L), magnesium salts should be administered for hypomagnesemia. If patients don't experience seizures or other serious symptoms of hypomagnesemia, give them oral magnesium salts; if they do, give them 2 to 4 g of magnesium sulphate intravenously over 5 to 10 minutes. Additionally, it is linked to higher mortality and longer hospital stays.
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Answer:
The correct answer is E. An elevated level of aldosterone makes both an increase in blood pressure and an increase in stored sodium ion.
Explanation:
Aldosterone, a hormone produced and secreted by the adrenal glands, transmits signals to the kidneys to retain more sodium and eliminate more potassium.
Hyperaldosteronism is a metabolic disorder characterized by an overproduction and secretion of the hormone aldosterone by the adrenal glands, which leads to decreased levels of potassium in the blood plasma and, in many cases to arterial hypertension. Having hyperaldosteronism, the adrenal glands produce too much aldosterone, which causes loss of potassium and sodium retention.