Answer:
The peripheral nervous system (PNS) is one of the two components of the nervous system, the other part is the central nervous system (CNS). The PNS consists of the nerves and ganglia outside the brain and spinal cord.[1] The main function of the PNS is to connect the CNS to the limbs and organs, essentially serving as a relay between the brain and spinal cord and the rest of the body.[2] Unlike the CNS, the PNS is not protected by the vertebral column and skull, or by the blood–brain barrier, which leaves it exposed to toxins and mechanical injuries. The peripheral nervous system is divided into the somatic nervous system and the autonomic nervous system. In the somatic nervous system, the cranial nerves are part of the PNS with the exception of the optic nerve(cranial nerve II), along with the retina. The second cranial nerve is not a true peripheral nerve but a tract of the diencephalon.[3]Cranial nerve ganglia originated in the CNS. However, the remaining ten cranial nerve axons extend beyond the brain and are therefore considered part of the PNS.[4] The autonomic nervous system is an involuntary control of smooth muscle and glands. The connection between CNS and organs allows the system to be in two different functional states: sympathetic and parasympathetic
Explanation:
The coenzyme required is pyridoxal phosphate. It is a vitamins B6 cofactor required for transamination reaction.
The left hemisphere of the cerebrum controls the ability to move and control your right hand
Answer:
They use a gene modification technique called CRIPSR-CAS9. The pigs have been given a gene that allows them to better regulate their own body temperature, whereby they burn fat, or at which animals consume their own fat.
Explanation:
British and Chinese scientists explained that they conducted the study by adding a mouse version of the UCP1 gene used for porcine embryos, which is used to regulate body temperature characteristic of most mammals, but not ordinary pigs.
It is problematic for pigs to maintain a stable body temperature, and this is especially complicated in pigs that are cold in the winter months because they have not been able to "store" fat.
Inserting the UCP2 gene could solve this problem.
