There are fewer antifungal, anti protozoan, and antihelminthic drugs compared to antibacterial drugs because these cells can be similar to human cells and therefore they can not achieve selective toxicity.
<h3>What is drug selective toxicity?</h3>
The expression 'drug selective toxicity' makes reference to the ability of a specific medication to discriminate between cells of different organisms (in this case human cells and other types of eukaryotic cells).
Drug selective toxicity is a fundamental issue during drug discovery and drug development.
In conclusion, there are fewer antifungal, anti protozoan, and antihelminthic drugs compared to antibacterial drugs because these cells can be similar to human cells and therefore they can not achieve selective toxicity.
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Neisseria gonorrhoeae is treated with a combination of drugs because the organism is resistant to many, including penicillin and fluoroquinolones.
Gonorrhoeae is a contender to produce an incurable illness due to the development or acquisition of resistance mechanisms for sulfonamides, penicillins, tetracyclines, ciprofloxacin, and more recently azithromycin and ceftriaxone during the past 80 years.
The Neisseria gonorrhoeae bacteria is the source of the sexually transmitted illness (STD) known as gonorrhea. The urethra in both men and women as well as the cervix, uterus, and fallopian tubes in women are all affected by N. gonorrhoeae infection.
Penicillin is the first drug that has been shown to be effective against syphilis and gonorrhea. However, compared to the dosage required to treat syphilis, which is often approximately 1,000,000 units, the drug used to treat gonorrhea is frequently administered in doses of just around 100,000 units.
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