Answer:
The interaction between cellulose-digesting organism in the gut of a termite and the termite is +
Explanation:
This relationship that exists between termites and the organisms present in their gut that are capable of degrading cellulose is a relationship called mutualism. Mutualism is part of the biological interactions in which both organisms benefit from the coexistence of both. For this reason this relationship would be positive.
The food of termites is wood, but they cannot produce the enzymes they would need for degradation of the wood. Thanks to this, there are small organisms called protozoa that are found in the intestine of the termites that allow them to degrade the cellulose present in the wood, these organisms help them to degrade the cellulose that by themselves they could not do since termites do not contain the cellulose enzymes responsible for cellulose degradation .In this way TERMITES can get what they need to perform its metabolism and biological functions.
Answer: Mutations in the GALT, GALK1, and GALE genes cause galactosemia.
Explanation: Galactosemia is a disorder that affects how the body processes a simple sugar called galactose. A small amount of galactose is present in many foods. It is primarily part of a larger sugar called lactose, which is found in all dairy products and many baby formulas. The signs and symptoms of galactosemia result from an inability to use galactose to produce energy.Classic galactosemia, also known as type I, is the most common and most severe form of the condition. If infants with classic galactosemia are not treated promptly with a low-galactose diet, life-threatening complications appear within a few days after birth. Affected infants typically develop feeding difficulties, a lack of energy (lethargy), a failure to gain weight and grow as expected (failure to thrive), yellowing of the skin and whites of the eyes (jaundice), liver damage, and abnormal bleeding. Other serious complications of this condition can include overwhelming bacterial infections (sepsis) and shock. Affected children are also at increased risk of delayed development, clouding of the lens of the eye (cataract), speech difficulties, and intellectual disability. Females with classic galactosemia may develop reproductive problems caused by an early loss of function of the ovaries (premature ovarian insufficiency). Galactosemia type II (also called galactokinase deficiency) and type III (also called galactose epimerase deficiency) cause different patterns of signs and symptoms. Galactosemia type II causes fewer medical problems than the classic type. Affected infants develop cataracts but otherwise experience few long-term complications. The signs and symptoms of galactosemia type III vary from mild to severe and can include cataracts, delayed growth and development, intellectual disability, liver disease, and kidney problems.
The answer is C i believe
