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Radda [10]
2 years ago
13

A male deer (stag) has long and heavy antlers while the female (doe) does not, Explain the purpose of this genetic variation and

why it can be a disadvantage?
Biology
1 answer:
Liono4ka [1.6K]2 years ago
8 0

Antlers are generally only found on male deer in other species of deer. Females with higher-than-normal testosterone levels, on the other hand, can grow antlers.

<h3>Why Do Deer Grow Antlers?</h3>

Antlers are grown by male deer to lure female deer for mating. Females will be shown a display as the antlers are developing during mating season, with each male attempting to become the most dominating.

Deer utilize their antlers to protect themselves from predators and other deer. When deer attack each other, their antlers might lock together, forcing the deer to starve to death.

For more information regarding antlers, visit:

brainly.com/question/17295060

#SPJ1

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Alleles positioned on the same chromosome are not always inherited together because during meiosis linked genes can became separate genes.

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Based on your results in Part A, which of the following statements most appropriately reflects the relative effectiveness of the
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Part A:

Treatment, Dose, Log of number of colonies, Mean.

Control -- 9.0, 9.5, 9.0, 8.9 9.1

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5.0 5.3, 5.9, 4.7 5.3

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which of the following statements most appropriately reflects the relative effectiveness of the vancomycin treatment, teixobactin treatment, and the control?

a) Teixobactin was significantly less effective than the vancomycin at reducing the number of MRSA colonies relative to the control.

b) Neither teixobactin or vancomycin were effective at reducing the number of MRSA colonies relative to the control.

c)Teixobactin was significantly more effective than the vancomycin at reducing the number of MRSA colonies relative to the control.

d) Teixobactin reduced the number of MRSA colonies about as effectively as did vancomycin relative to the control

Answer: A. Teixobactin was significantly less effective than the vancomycin at reducing the number of MRSA colonies relative to the control.

Explanation: By way of definition, Methicillin-resistant Staphylococcus aureus (MRSA) is a significant cause of health care-associated infections. Vancomycin remains an acceptable treatment option. Though there is an evolution of microbial resistance to vancomycin which posed to be a problem, in particular, within healthcare facilities such as hospitals. While newer alternatives to vancomycin exist, the widespread use of vancomycin makes resistance to the drug a significant worry, especially for individual patients if resistant infections are not quickly identified and the patient continues the ineffective treatment. There has been a welcome increase in the number of agents available for the treatment of MRSA infection for instance the Teixobactin, Linezolid, daptomycin, telavancin and ceftaroline. These drugs have certain differentiating attributes and may offer some advantages over vancomycin, but they also have significant limitations.

Teixobactin was also found to be effective in vivo, when used to treat mice infected with methicillin-resistant S. aureus (MRSA), and Streptococcus pneumoniae. The dose required to achieve 50% survival against MRSA is only 10% of the PD50 dose of vancomycin, an antibiotic typically used for MRSA. However, Teixobactin was reported to be potent in vitro against all gram-positive bacteria tested, including Staphylococcus aureus and difficult-to-treat enterococci, with Clostridium difficile and Bacillus anthracis being exceptionally vulnerable. It also killed Mycobacterium tuberculosis.

Teixobactin is not active against bacteria with an outer membrane such as gram negative pathogens, particularly carbapenem resistant enterobacteriaceae. It is possible from the results, that genes encoding resistance to teixobactin are already present in soil bacteria. Resistance could also arise by mutation after prolonged use of teixobactin in patients. So based on the experiment conducted, Teixobactin was significantly less effective than the vancomycin at reducing the number of MRSA colonies relative to the control.

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Secondly, pollution from the power plants in the area- consisting mainly of the toxic chemical mercury- pose risk to anglers in the area. These contaminants can bind to sediment and enter bodies of small, bottom-dwelling organisms like worms, clams or crustaceans. Through a process known as bioaccumulation, fish that consume contaminated organisms can accumulate these toxins in their own tissue. Anglers are warned to limit their consumption of certain fish from the Bay due to potentially harmful levels of the chemical.

Lastly, exposure to air pollution will affect those living in, and visiting, the Bay. Air pollution can also cause headaches, dizziness, and nausea, as well as long-term health effects including heart disease, lung cancer, and respiratory diseases such as emphysema. Air pollution can also cause long-term damage to people's nerves, brain, kidneys, liver, and other organs

3. The Chesapeake Bay is arguably the most studied large body of water on earth. Forty years of intense scientific investigation by leading estuarine scientists have identified why the Chesapeake is degraded and how to fix it. No other water body in the world can boast this level of scientific understanding.

The first action being taken to protect the bay from air pollution is by planting more plant life in the area. Through a process called attenuation, forests can reduce the amount of pollutants in the air. The plants roots and leaves and forest soils absorb and trap airborne pollutants. They can capture more than 85 percent of the nitrogen that falls onto them from the air, keeping it out of the bay.

Secondly people are changing the way they travel around and near the Bay. To lower air pollution people consider learning how to drive the Bay-friendly way; walk bike, take public transport and carpool when possible. They also try to use electric or manual lawn mowers and yard tools instead of gas-powered machines.

Thirdly, the Environmental protection agency developed the Chesapeake Bay Total Maximum Daily Load in 2010 (TMDL). It set limits on the amount of nitrogen, phosphorus, and sediment that can enter the Bay and its tidal rivers to meet water quality goals. Ever since, the EPA has kept track on the progress of the Chesapeake Bays health.

4.

In planting more trees and plants near the Chesapeake Bay area, it will prevent some sediment and pollutants entering the water. The plants roots and leaves and forest soils absorb and trap airborne pollutants. They can capture more than 85 percent of the nitrogen that falls onto them from the air, keeping it out of the bay through attenuation.

Secondly by reducing the amount of pollutants entering the air via petrol powered machines and cars, the amount of pollutants entering the waters will decrease. Each year, more than 85 million pounds of nitrogen pollution—about one-third of the Bay's total yearly load—comes from air pollution, and with 18 million people living in the Chesapeake Bay watershed, you can imagine how much difference cutting down on transport generated pollution would make.

Finally setting the TMDL will aid in preventing the amount of pollutants in the water reaching a high concentration and hopefully reduce the amount of algal blooms and dead zones within the bay.

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