The answer is epididymis.
<span>Testicles contain seminiferous tubules, in which sperm is produced, and an epididymis, in which sperm matures. Mature sperm passes through the epididymis to the vas deferens. There, it combines with a liquid secretion from the seminal vesicles and continues through the prostate gland to the urethra. Sperm containing fluid is called semen and it is ejected through the urethra.</span>
<span>There are numerous proteins in muscle. The main two are thin actin filaments and thick myosin filaments. Thin filaments form a scaffold that thick filaments crawl up. There are many regulatory proteins such as troponin I, troponin C, and tropomyosin. There are also proteins that stabilize the cells and anchor the filaments to other cellular structures. A prime example of this is dystrophin. This protein is thought to stabilize the cell membrane during contraction and prevent it from breaking. Those who lack completely lack dystrophin have a disorder known as Duchene muscular dystrophy. This disease is characterized by muscle wasting begininng in at a young age and usually results in death by the mid 20s. The sarcomere is the repeating unit of skeletal muscle.
Muscle cells contract by interactions of myosin heads on thick filament with actin monomers on thin filament. The myosin heads bind tightly to actin monomers until ATP binds to the myosin. This causes the release of the myosin head, which subsequently swings foward and associates with an actin monomer further up the thin filament. Hydrolysis and of ATP and the release of ADP and a phosphate allows the mysosin head to pull the thick filament up the thin filament. There are roughly 500 myosin heads on each thick filament and when they repeatedly move up the thin filament, the muscle contracts. There are many regulatory proteins of this contraction. For example, troponin I, troponin C, and tropomyosin form a regulatory switch that blocks myosin heads from binding to actin monomers until a nerve impulse stimulates an influx of calcium. This causes the switch to allow the myosin to bind to the actin and allows the muscle to contract. </span><span>
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Answer: 1/4
Explanation:
Firstly, in order for both parents to be type A and have children with type o blood, their blood types must both be Ao. Since o is a recessive blood type, a punnett square shows there is a 25% chance any child of theirs will have type o blood. If neither parent is color blind and they have a son who is, it implies that the mother is a carrier of colorblindness and has the genotype XᴮXᵇ. If you do a punnett square of the not colorblind father (XᴮY) and the mother, it shows that a daughter would have a 0% chance of being colorblind. Therefore colorblindess is irrelevent, since there is no possibility of the daughter not having normal color vision. In conclusion, there's a 25% chance she will have type o blood and not be colorblind, since the other 75% chance would be having type A blood and not being colorblind.
The nurse can give the following instructions:
1. the procedure will most likely last for ten minutes
2. since it is still an infant, the child will be put under anaesthesia
3. a pulsed dye laser treatment will be given
4. if general anaesthesia will be given, then there are special rules for eating and drinking restrictions before procedure
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Answer:
7.false 8.commensalism 9.last one
Explanation:
