<span>After staring at a blue light for a few minutes, Yoko shifts her gaze to a white wall and experiences an afterimage in the color yellow. Yoko's experience provides support for the opponent-process theory of color vision.
The opponent-process theory is proposed by
</span>color vision. This model was first proposed in 1878 by Ewald Hering and <span>states that when one emotion (in this case : color) is experienced, the other is suppressed. In Yoko's case she experiences (looked) the blue color long enough to supres the white color,</span>
Answer: a blacktop highway
Sorry for a late answer... But here we go!
https://www.sciencelearn.org.nz/resources/102-methods-of-pollination
Answer:
No, there are multiple ways in which different mutations in the same gene can cause the same phenotype
Explanation:
Several different mechanisms of mutation can lead to the same phenotype. For example, lets say our phenotype is that flies have white eyes, and we know that this occurs in one particular gene that normally makes the eye colour red. (the red gene)
These mutations likely rendered the red gene ineffective (as the eyes are not red). However, this could happen in a variety of ways.
- There could be a single base deletion in the first exon of the mRNA, changing the reading frame of the protein and messing up the entire sequence (a frame shift mutations)
- The entire gene could be deleted
- A single base could be substituted in an important site of the gene, for example, one which translates into a catalytic residue or binding site in the protein
- There could be an inversion at the promoter region of the gene, such that a transcription factor can no longer bind to transcribe the gene.
There are countless other ways in which a mutation could have been caused. Therefore, just because we know the same gene is affected does not mean that we can assume the mutations are identical.
Since the aphotic zone does not recieve sunlight it relys on the photic zone for the sunlight which in terms it does penetrate.
