If a particular job of a cell was to swim a long distance to an egg with a flagella, then it would need more mitochondria. As mitochondria undergo cellular respiration and essentially utilize the carbohydrates for that particular cell to produce the final product of ATP high energy containing molecules needed for the movement.
Muscle spasms<span>Hypocalcemia can cause
emotional disturbances
depression of the nervous system
muscle weakness
sluggish reflexes
muscle spasms</span>
Is compatable to defection or malfunction
There should be options for this question. I manged to find them elsewhere. They are:
A) the period during which tremendous growth occurs and the organs continue to develop and become functional
B) the period during which the umbilical cord develops
C) the period during which the major organs and structures of the organism first develop
D) the period during which the zygote moves down to the uterus and begins to implant in the lining
The correct answer is C. The embryonic period is the period during which the major organs and structures of the organism first develop. The embryonic period lasts from implantation of the egg in the uterus until about 8 weeks from the time of conception. It is the second major stage of prenatal development, prior to embryonic is the germinal stage and lastly is the fetal stage.
The correct answer is the last statement.
If the regulatory serine is mutated to alanine, then acetyl-CoA carboxylase will get activated spontaneously and will produce malonyl-CoA. The increased concentrations of malonyl-CoA will obstruct the oxidation of fatty acids by preventing the entry of fatty acids into the mitochondria.
It is because the AMP-activated protein kinase phosphorylates the serine residues of acetyl-CoA carboxylase to inactivate it. If a mutation occurs in such residues, then the AMPL cannot phosphorylate acetyl-CoA carboxylase and this enzyme will get activated spontaneously.
In such a situation, there will be more than sufficient production of malonyl-CoA, which will inhibit the admittance of more fatty acid getting inside the mitochondria; this will indirectly prevent the oxidation of fatty acids.
