<span>well this is an interesting question and i would say it may depend on what type of cancer cells you are growing and what type of "normal" cells your growing. One possibility is that cell fusion events may occur between your cancer cells and normal cells, thus creating a few options 1 - making the normal cell cancerous, 2 - making the cancer cell that fused with the normal cell not cancerous anymore. 3 - either way the fused cell will have a different genotype and hence be a different cell.</span>
The correct answer is choice B, Tt. I hope this helps.
Answer:
In human vision, the cone visual opsins are grouped into four photoreceptor protein families LWS, SWS1, SWS2, RH2
.
- SWS1: produce pigments sensitive to very short wavelengths, UV-violet, 360-450 nm.
- SWS2: produce pigments sensitive to short wavelengths, blue, 450-495 nm
- RH2: produce pigments sensitive to medium wavelengths, green, 495-560 nm
- LWS: produce pigments sensitive to long wavelengths, yellow-red, 560-770nm.
Explanation:
Photoreceptor proteins are light-sensitive proteins that mediate light-induced signal transduction, thus they are involved in the sensing and response to light in a variety of organisms.
The photoreceptor proteins are classified based on the chemical structure of the chromophores involved, the light absorption and on the protein sequence.
This photoreceptor proteins are located at the cone photoreceptor cells and are responsible of photopic vision.
For scotopic vision, rhodopsin is responsible. Rhodopsins are the visual pigments (visual purple) of the rod photoreceptor cell in the retina. They are responsible of human vision in dim light, as it contains a sensory protein that converts light into an electric signal.
Answer:
Mutations in introns create alternative splicing site which in turn make mRNA with reduced or no translation.
Explanation:
Introns regulate the splicing of heterogenous RNA during post transcriptional modifications as well as affect the stability of mature mRNA. The stable mRNA is more likely to be translated into proteins. So, introns also regulate the translation of mRNA.
In human beta thalassemia, mutations in intron create alternative splicing sites which in turn affect the formation of beta globin chain of hemoglobin. Lack of beta globin chain reduces the amount of functional hemoglobin and causes anemia.
I'm really not sure but i think that its law because look at the LAW of gravity and it shows how gravity pretty much works
