Answer:
D
Explanation:
all living things are made up of many cells
Answer:
The changes in the sequence of nucleotides present within a promoter is a prime cause of the defected transcriptional regulation, which may eventually result in disease. However, not every modification within the sequence of a promoter influences the regulation of transcription, it relies upon the nature and the location of the genetic defect.
When a mutation results within the sequence of a promoter region it may hamper the usual procedures of gene stimulation by affecting the step by step alignment of the transcription factors at the promoter region. Therefore, as a consequence, a mutation within the sequence of a promoter may result in the enhancement or reduction in the level of mRNA and thus protein.
C but it's still not exactly correct, they would just be definite carriers of the colour blind trait but only the girls are able to obtain the gene
The answer is C. It eliminates the toxic chemical problem. Hope this helps!
Ti plasmid encoded octopine and nopaline catabolism in Agrobac terium Ti plasmid-encoded genes required by the micro organism for opine catabolism.
The occ and noc areas in octopine and nopaline Ti plasmids, respectively, are accountable for the catabolism of octopine and nopaline in Agrobacterium. The functions are activated within the presence of the opines with the aid of OccR and NocR, related regulatory proteins, and the promoters incorporate commonplace collection motifs.
we have investigated Ti plasmid in heterologous interactions among the regulators and the promoters. previous experiments the usage of all possible heterologous combos of opines, regulators, and promoters in vivo had demonstrated that handiest the aggregate of nopalme, NocR, and the occ promoter led to restricted promoter activation. We now display that OccR and NocR bind to the heterologous promoters in vitro and in vivo.
The weak or non-existent promoter activation truly located can be explained by the idea that OccR and NocR use distinct activation mechanisms; we investigated protein-brought about DNA bending due to reports that the two regulators vary in this respect.
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