luconeogenesis is a ubiquitous process, present in plants, animals, fungi, bacteria, and other microorganisms.[2] In vertebrates, gluconeogenesis takes place mainly in the liver and, to a lesser extent, in the cortex of the kidneys. In ruminants, this tends to be a continuous process.[3] In many other animals, the process occurs during periods of fasting, starvation, low-carbohydrate diets, or intense exercise. The process is highly endergonic until it is coupled to the hydrolysis of ATP or GTP, effectively making the process exergonic. For example, the pathway leading from pyruvate to glucose-6-phosphate requires 4 molecules of ATP and 2 molecules of GTP to proceed spontaneously. Gluconeogenesis is often associated with ketosis. Gluconeogenesis is also a target of therapy for type 2 diabetes, such as the antidiabetic drug, metformin, which inhibits glucose formation and stimulates glucose uptake by cells.[4] In ruminants, because dietary carbohydrates tend to be metabolized by rumen organisms, gluconeogenesis occurs regardless of fasting, low-carbohydrate diets, exercise, etc.[5]
The correct answer is: E) cleavage furrow formation and cytokinesis
Cytochalasin B (cytos-cell and chalasis-relaxation) is a molecule which inhibit network formation by actin filaments by blocking monomer addition. As a result, itshortens actin filaments. This molecule is involved in cytoplasmic division where it blocks the formation of contractile microfilaments. One of the microfilament’s function includes cytokinesis and formation of cleavage furrow so these functions are affected by cytochalasin B.
I would say C.
I hope it’s right or u can individually search up each of those animals to check that they’re a secondary consumer.
Monosaccharides are made of carbon,hydrogen, and oxygen atoms
