(C) Autoimmune destruction of B-cells of the pancreas is the etiological cause of this patient's symptoms.
The death of insulin-producing pancreatic beta-cells by autoreactive T cells characterizes type 1 diabetes (T1D), an autoimmune condition. Patients eventually need lifetime insulin treatment to maintain normal glycemic control when beta-cell loss causes insulin insufficiency and hyperglycemia.
Pancreatic B-cell autoimmunity is the cause of type 1 diabetes mellitus. Hyperglycemia, polyuria, increased thirst, weight loss, increased hunger, and nausea/vomiting are a few of the typical signs and symptoms of type 1 diabetes.
Elevated blood sugar levels and possible glucose present in the urine Along with other symptoms, diabetic ketoacidosis (as in this patient) might show up as ketone bodies in the urine and Kussmaul breathing.
Here is another question with an answer similar to this about type 1 diabetes: brainly.com/question/14823945
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Question correction:
A 12-year-old girl becomes comatose and is rushed to the hospital by her parents. She went to school feeling ill 2 days before the admission. She vomited that evening. Her vomiting persisted with only an 8-hour pause during sleep. She is breathing deeply and rapidly; her breath has a fruity odor. Her parents mention that her appetite has increased. She has also been drinking a lot of fluids; subsequently, she has been urinating more than normal. Urinalysis reveals 3+ glucose levels and 2+ ketone bodies. What is the etiological cause of this patient's symptoms?
A. Insulin resistance
B. Increase in counterregulatory hormones
C. Autoimmune destruction of B-cells of the pancreas
D. Post-Epstein-Barr virus infection
E. Autoimmune destruction of pancreatic acini cells
Answer:
True.
Explanation:
Friction may be defined as the force that oppose the motion of the one object moving relative to the another object. Friction is a type of non conservative force.
Higher coefficient of friction means that friction is less. Less friction has the higher tendency to slide. Oiling and greasing reduces the friction of object and makes it more slippery.
Thus, the answer is true.
Answer:
The correct answer will be:
1. Acetylcholine binds to receptors on the motor end plate
2. Chemically-regulated ion channels open, causing depolarization
3 End plate potentials trigger action potential(s).
4. Transverse tubules convey potentials into the interior of the cell
5. Ca++ is released from the sarcoplasmic reticulum
6. Ca++ binds to troponin, pulling on tropomyosin
7. Binding sites on actin are uncovered, allowing myosin to bind and carry out power strokes
8. Force increases.
9. Ca++ is pumped (re-sequestered) into the sarcoplasmic reticulum
10. Force decreases.
Explanation:
The muscle contraction is a highly controlled mechanism which begins at the neuromuscular junction with the release of the acetylcholine neurotransmitter. This neurotransmitter causes the depolarization of the membrane by binding to the receptors of the motor end plate which generates an action potential. This action potential is transmitted via T-tubules from sarcolemma to the sarcoplasmic reticulum.
The sarcoplasmic reticulum releases calcium ions which binds to the troponin protein. This troponin removes the protein tropomyosin from the actin causing the rotation of the tropomyosin exposing the binding sites for myosin. The myosin binds to the actin using energy from the ATP which pulls the actin causing contraction. Another ATP binds the myosin head which weakens the bond between myosin and actin which releases the myosin which decreases the force between them decreases and the muscles relax.
I don’t know but good luck finding the answer
Answer:
0.2 ml
Explanation:
Stock dilution is done by using the formula C1V1 = C2V2 where,
C1V1 = Start volume and concentration
C2V2 = Final volume and concentration
Here,
C1V1 = C2V2
5 mg/ml * V1 = 0.1 mg/ml * 10 ml
V1 = ( 0.1 * 10 ) / 5 ml
= 1/5 ml
= 0.2 ml
So 0.2 ml of nitroglycerin stock is needed to make the final solution.