Answer:
This is achieved because the sodium channels have a refractory period following activation, during which they cannot open again. This ensures that the action potential is propagated in a specific direction along the axon.
Answer:
As, this individual is expending fructose syrup.
Fructose is changed over to purine which is additionally changed over to uric acid.
Along these lines, more fructose in the eating routine then more purines in the body which will additionally frame progressively uric acid.
This uric acid gathers and causes gout.
What is the best clarification for the introduction of gout right now please clarify why different decisions are not reliable with the information or what extra data you may need to choose the underlying driver?
1. overproduction of purines-They are not being created in the body however yes they are certainly are delivered from the abundance fructose expended.
2. diminished rescue of purines-No, this isn't the main driver.
3. diminished urinary discharge of uric acid this can occur as less uric acid is being discharged in the pee.
When, contrasted with the ordinary individual the patient is discharging less uric acid.
Along these lines, third point is the purpose behind gathering of uric acid in the body.
Answer:
Atelectasis is a condition in which all or part of a lung becomes airless and collapses. Blockage of the bronchial tubes is a common cause of atelectasis. Shortness of breath can develop if oxygen levels are low or pneumonia occurs.
HOPE THIS HELPED!!!!!! XDDD
Answer:
The truth about the hormone leptin and obesity. It's been called the " obesity hormone" or "fat hormone" -- but also the "starvation hormone." When scientists discovered leptin in 1994, excitement arose about its potential as a blockbuster weight loss treatment. Even today, the Internet is loaded with sites that sell leptin supplements.
Explanation:
