Pioneer species are the first species that colonize places where there were previously no other living organisms, such as algae that colonize bare rocks. ... Therefore, they pave the way for other species to establish themselves at the location through the creation of new potential ecological<span> niches.</span>
Answer: B
Explanation:
Mitosis produces two diploid (2n) somatic cells that are genetically identical to each other and the original parent cell, whereas meiosis produces four haploid (n) gametes that are genetically unique from each other and the original parent (germ) cell.
ADH and aldosterone secretion are part of the body's compensation for A serious hemorrhage. Vasopressin or ADH is a hormone synthesized as a peptide prohormone in neurons in the hypothalamus and is converted to AVP. The primary role is maintaining osmolarity and thus maintaining the volume of water in the extracellular fluid, and the secondary function of ADH is raising blood pressure by constricting blood vessels. Aldosterone on the other hand is essential for sodium conservation in the kidney.
Answer:
α-1,6-glycosidic linkage.
Explanation:
The carbohydrates are the polymers made of the sugars and bonded together through the glycosisdic bonds. The monosaccharides are the simplest unit of the carbohydrates.
The glycogen and amylopectin share similar structural features. Both have α-1,4-glycosidic linkage in their linear structure. The branching has been introduced in the glycogen and amylopectin structure through the α-1,6-glycosidic linkage.
Thus, the answer is α-1,6-glycosidic linkage.
When a DNA sequence alteration results in a stop codon rather than a codon that specifies an amino acid, it is known as a nonsense mutation. This is rarely seen in 10% of patients with genetic disease.
<h3>What is nonsense mutation?</h3>
A nonsense mutation in a DNA sequence causes a premature stop codon, also known as a nonsense codon, in the transcribed mRNA as well as a shortened, ineffective, and typically nonfunctional protein product.
Because stop codons, also known as nonsense codons, signal the completion of protein synthesis rather than encoding for an amino acid, they are the source of the term "nonsense mutation."
Examples of illnesses for which nonsense mutations have been implicated as contributing factors include: Cystic fibrosis (produced by the G542X mutation in the cystic fibrosis transmembrane conductance regulator); (CFTR) Beta-globin (thalassemia) Hurler disease.
Learn more about nonsense mutation, here:
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