Answer: a. dead organisms from the marine food web.
c. liberation through ATP hydrolysis in living organisms.
Upwelling is a wind driven motion of lower bottom nutrient rich and warmer water on the surface of the water body. This wind driven motion facilitates the movement of nutrients available for growth of primary producers like phytoplanktons growing on the surface of water body. The dead organisms from the marine food web get decomposed and the organic matter obtain after decomposition is a rich source of phosphorous. This phosphorous gets transferred to the upper layers of the water body by upwelling. In aquatic organisms ATP hydrolysis occurs which is a catabolic process that uses water to split the bonds present in the ATP molecule and hence, releases energy for functions performed by them along with a release in phosphate atom. This phosphate gets mixed with the water. Therefore, PO32 come from dead organisms from the marine food web and liberation through ATP hydrolysis in living organisms that circulates due to upwelling.
Answer:
The correct answer is 4: "The exception to Mendelian laws of inheritance that best explains the mentioned symptoms is codominance".
Explanation:
In codominance, both alleles can be expressed. In these cases, heterozygote individuals<em> instead</em> of showing an <em>intermediate phenotype</em>, express both of the alleles. Their phenotype is an additive expression of their parents' genes.
In cystic fibrosis, there is a gene responsible for coding for a protein named "cystic fibrosis transmembrane conductance regulator, CFTR".
-Most of the people have two copies of the normal allele and produce the functional CFTR protein form.
-Patients with cystic fibrosis have two copies of the mutated allele and so produce the mutated and dysfunctional form for this protein.
-Heterozygote people possess only one normal CFTR allele and a mutated form for the same allele and produce a normal protein and a mutated protein.
In the last case, both alleles are codominant and they express in heterozygote individuals. Given the fact that the normal allele produces enough functional CFTR protein, these individuals do not have any adverse effect and the mutated allele is recessive at a physiological level.
Other digestive diseases include: ... Intestinal problems, such as polyps and cancer, infections, celiac disease, Crohn disease, ulcerative colitis, diverticulitis, malabsorption, short bowel syndrome, and intestinal ischemia. Gastroesophageal reflux disease (GERD), peptic ulcer disease, and hiatal hernia.
