When a DNA sequence alteration results in a stop codon rather than a codon that specifies an amino acid, it is known as a nonsense mutation. This is rarely seen in 10% of patients with genetic disease.
<h3>What is nonsense mutation?</h3>
A nonsense mutation in a DNA sequence causes a premature stop codon, also known as a nonsense codon, in the transcribed mRNA as well as a shortened, ineffective, and typically nonfunctional protein product.
Because stop codons, also known as nonsense codons, signal the completion of protein synthesis rather than encoding for an amino acid, they are the source of the term "nonsense mutation."
Examples of illnesses for which nonsense mutations have been implicated as contributing factors include: Cystic fibrosis (produced by the G542X mutation in the cystic fibrosis transmembrane conductance regulator); (CFTR) Beta-globin (thalassemia) Hurler disease.
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MRNA, ribsomes,tRNAs, and many enzymatic factors
Augustinian friar Gregor<span> Johann </span><span>Mendel.
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Because it is polar and it is to large to cross the membrane and because the concentration gradients are to strong so they have to be diffused.
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Well for starters, they do not have leaves which reduces transpiration. They also grow really long roots that can absorb the smallest traces of moisture in the earth.
