I think its like the boss or the person who asigns docters to paitants
C.Ecstasy
Ecstasy <span>is a synthetic drug that alters mood and perception. It is chemically similar to both stimulants and hallucinogens by stimulating increased energy and pleasure. Along with creating a distorted sense of sensory and time perception.</span>
Ethos means attribute of spirit of a era, culture, or community. Some synonyms include: <span><span>spirit, character, atmosphere, climate, mood, feeling, tenor, or essence. Hope this helped you!</span></span>
Answer:
Octavio she will dance with him after her one dance with Myron as he has asked first
Explanation:
This option would be the least likely to cause a ruckus or conflict.
(btw, who says chivalry is dead?)
Answer:
Multiple sclerosis (MS) is the most common demyelinating and an autoimmune disease of the central nervous system characterized by immune-mediated myelin and axonal damage, and chronic axonal loss attributable to the absence of myelin sheaths. T cell subsets (Th1, Th2, Th17, CD8+, NKT, CD4+CD25+ T regulatory cells) and B cells are involved in this disorder, thus new MS therapies seek damage prevention by resetting multiple components of the immune system. The currently approved therapies are immunoregulatory and reduce the number and rate of lesion formation but are only partially effective. This review summarizes current understanding of the processes at issue: myelination, demyelination and remyelination—with emphasis upon myelin composition/architecture and oligodendrocyte maturation and differentiation. The translational options target oligodendrocyte protection and myelin repair in animal models and assess their relevance in human. Remyelination may be enhanced by signals that promote myelin formation and repair. The crucial question of why remyelination fails is approached is several ways by examining the role in remyelination of available MS medications and avenues being actively pursued to promote remyelination including: (i) cytokine-based immune-intervention (targeting calpain inhibition), (ii) antigen-based immunomodulation (targeting glycolipid-reactive iNKT cells and sphingoid mediated inflammation) and (iii) recombinant monoclonal antibodies-induced remyelination.Keywords: calpain, central nervous system, demyelination, fingolimod, glycolipids, lipids, multiple sclerosis, myelin, myelination, NKT cells, oligodendrocytes, remyelination, T cells
Explanation:
