True because carbohydrates are in the cell membrane.
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The correct answer is that mutant cells will exhibit diminished oxygen consumption; decreased glycolysis results in decreased Kreb's cycle and electron transport chain.
The PFK2 enzyme catalyzes the generation of F26BP, this binds with the allosteric site of PFK-1 and increases the affinity of PFK-1 with F6P and also decreases the affinity of allosteric inhibitors citrate and ATP to PFK-1. Thus, PFK-1 will combine with F6P at a greater rate.
This ultimately results in more glycolysis, thus, more ETC and more consumption of O2. If there is no PFK2, then there will be a reduction in glycolysis, TCA, ETC, and consumption of oxygen.
The PFK2 is an enzyme accountable for monitoring the rates of gluconeogenesis and glycolysis in the human body. In the absence of glycolysis, there will be a reduction in TCA, ETC, and consumption of O2.
The hydrogen bonding of nucleotides into a new DNA molecule is facilitated by enzymes. Hydrogen bonding is the electromagnetic attraction between polar molecules in which hydrogen is bound to a larger atom, such as oxygen or nitrogen. The nucleotides in a base pair are complementary which means their shape allows them to bond together with hydrogen bonds. The A-T pairs forms two hydrogen bonds while the C-G pair forms three. The enzymes called DNA polymerases join the nucleotides by way of phosphodiester bonds.
