Incoordination of musles during movement is known as ataxia.
This can be demonstrated by using an acellular environment to observe the 32P label entering phosphatidylethanolamine (PE) molecules by direct exchange.
Phosphatidylethanolamine (PE) is a major lipid component of cellular membranes in a wide range of organisms.
A scientist proposed that the 32P label was entering PE molecules by direct exchange, this can be proved using acellular environment.
In acellular environment, there aren't any bacterial cells to synthesize new PE. So if the 32P is incorporated into the PE in the acellular environment that means the proposed idea in the question stem is correct.
If no 32P is incorporated into PE in the acellular environment, then that means proposed idea is not correct.
Disclaimer: The question is not completed.
A scientist proposed that the 32P label was entering PE molecules by direct exchange (swapping phosphate groups with those found in solution) and NOT through synthesis of new PE by bacterial cells. What experimental modification can show this is NOT the case?
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Answer: False
Explanation:
A forcible breaking up of the adhesions in an anklosis to allow more mobility in the joint is known as Arthroclasia.
It is not related to the therapeutic procedure in which involves the placing of the ankylosed joint in traction.
This techniques provides mobility to the joint which is clogged by the fusion of bones and can cause abnormal stiffness and immobility.
