Answer:
Inhibit control over ions.
Explanation:
The immediate effect of the neurotoxin on the postsynaptic neuron is to inhibit control over ion concentrations across the cell membrane. The neurotoxin blocks nervous transmission at synapses which prevents the vesicles containing the neurotransmitter from fusing with the presynaptic membrane so the information does not transfer from one neuron to another and as a result the body is present at constant relaxation mode. The membrane potential of postsynaptic neuron will decrease due to neurotoxin.
Answer:
I thought it was cell division
Before the 1600's (when Taxonomy came into the world) many scientists divided organisms into two groups: plants and animals.
<span>The most serious complication that can appear in a client experiencing kidney failure is fluid retention. This could lead to swelling in your arms and legs or fluid in your lungs wich can cause a pulmonary edema so a client can happen a sudden death if not treated accordingly.</span>
Answer:
Thioredoxin
Explanation:
Ribonucleotides serve as precursors for synthesis of deoxyribonucleotides and the reaction is catalyzed by enzyme ribonucleotide reductase. The reduction of a ribonucleotide into deoxyribonucleotide requires a pair of hydrogen atoms.
These hydrogen atoms are supplied from NADPH via ubiquitous protein thioredoxin. The oxidized form of thioredoxin is reduced back in a reaction catalyzed by thioredoxin reductase.
