Answer: hair color and height.
Explanation:
Answer:
Charles Darwin was the man who came with the theory of natural selection. He witnessed finches within the Galapagos Islands and made notes on the similarities and differences of the finches across the group of islands. Each of the islands comprises finches, which were identical, however distinct in different ways.
Darwin found that the finches appeared to vary on the basis of the food sources available on each of the islands. If the prime food were seeds, the finches seemed to possess thicker beaks in order to break the seeds so that they can consume them.
On the other hand, if the prime food sources were insects, then the finches seemed to exhibit smaller and pointer beaks so that they could hold the insects readily. In this way, there is unity in diversity. All of these birds are finches and exhibited a common ancestor from which they have originated into the distinct species as mentioned.
Their variations lie in the habitats, in which they now inhabit. They had to amend with their environments in order to thrive, thus, offering diversity to unity.
Enzymes are also known as Biological catalysts. An enzyme working at its maximum possible rate, where the substrate concentration is in excess is <span>V max.
</span><span>The reaction is slow because the molecules are moving slowly and they will not often collide with the active site of an enzyme, so the binding of an enzyme and substrate is rare.</span>
<span>Rhabdomyolysis constitutes a common cause of acute renal failure and presents paramount interest. A large variety of causes with different pathogenetic mechanisms can involve skeletal muscles resulting in rhabdomyolysis with or without acute renal failure. Crush syndrome, one of the most common causes of rhabdomyolysis presents increased clinical interest, particularly in areas often involved by earthquakes, such as Greece and Turkey. Drug abusers are another sensitive group of young patients prone to rhabdomyolysis, which attracts the clinical interest of a variety of medical specialties.
We herein review the evidence extracted from updated literature concerning the data related to pathogenetic mechanisms and pathophysiology as well as the management of this interesting syndrome.
Keywords: Rhabdomyolysis, acute renal failure, myoglobin, crush syndrome
The first case of the crush syndrome, which constitutes one of the main causes of rhabdomyolysis, was reported in Sicily in 1908, after an earthquake1,2. In 1930, in the Baltic area, an epidemic of myoglobinuria was observed due to consumption of contaminated fish. Interest in rhabdomyolysis and crash syndrome was stimulated during the World War II particularly after the bombing in London, where the victims developed acute renal failure and myoglobinuria1.
Rhabdomyolysis is a rupture (lysis) of skeletal muscles due to drugs, toxins, inherited disorders, infections, trauma and compression3. Lysis of muscle cells releases toxic intracellular components in the systemic circulation which leads to electrolyte disturbances, hypovolemia, metabolic acidocis, coagulation defects and acute renal failure due to myoglobin4.
The skeletal muscle consists of cylindrical myofibrils, which contain variant structural and contraction proteins. Actin and myosin, arranged in thin and thick filaments respectively, form the repeated functional units of contraction, the sarcomeres5. The sarcoplasmic reticulum constitutes an important cellular calcium storage. It is structurally connected to the t-tubules, that are formed by invaginations of the muscle cell plasma membrane, the sarcelemma, around every fibril (Figure 1). After the sarcelemma depolarization, the stimulation arrives, through the t-tubules junctions, at the sarcoplasmic reticulum, inducing the calcium ions release and triggering muscle contraction6.</span>
