The answer is C because there is no inference in the statement like the others.
Answer:
Hyperpolarization means that the membrane potential becomes more negative than the resting potential.
Explanation:
The voltage across the membrane of a neuron that is at rest and not sending out signals is called the resting membrane potential, or just the resting potential. The concentration gradients of ions across the membrane and the amount of each type of ion that can pass through the membrane determine the resting potential. When a neuron is at rest, there are different levels of sodium and potassium on both sides of the cell membrane. Ions move down their gradients through channels. This creates a difference in charge, which gives rise to the resting potential.
When the membrane potential at a particular location on the neuron's membrane gets more negative, this phenomenon is known as hyperpolarization. Depolarization, on the other hand, occurs when the membrane potential becomes less negative (more positive). Both depolarization and hyperpolarization can take place as a result of the opening and closing of ion channels in the membrane, which changes the rate at which certain types of ions can enter or leave the cell.
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<em>Your question is incomplete, but most probably your full question was </em><em>Hyperpolarization means that the membrane potential becomes Group of answer choices </em>
<em>more negative than the resting potential </em>
<em>more positive than the resting potential</em>
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The raw material from which sedimentary rocks are formed is D. Weathered remains of other rocks.
Water is essential to life because it creates us humans, plants, animals, etc. So, if we find water on Mars it would be important because water is essential to life, meaning we would know that there would be some kind of life on Mars because water makes up anything.
To find the functional connection's purpose in <em>A. thaliana's</em> chromatin, transcription, and splicing. It was studied and evaluated with the RNAPII processivity (involvement of TFIIS), chromatin structure (roles of BRM, SWI3c, and H1.3 in AS), and spliceosome formation were all investigated at three separate levels (characterization of the role of spliceosome disassembly factor NTR1 in the selection of splice sites).
The alteration of RNAPII elongation rate as the mechanism for NTR1 and TFIIS influences splicing.
Lack of NTR1 causes both localized lower RNAPII levels at those splice locations as well as defective splicing. This can be translated as quicker transcription elongation over those sites in accordance with the kinetic concept of transcription/splicing coupling.
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