D) Photosynthesis
Explanation:
Plants use Carbon dioxide in the process that turns sunlight, carbon dioxide and water into glucose and oxygen
Answer:
A blood test is used to determine whether the A and/or B characteristics are present in a blood sample. It is not possible to determine the exact genotype from a blood test result of either type A or type B. If someone has blood type A, they must have at least one copy of the A allele, but they could have two copies
Explanation:
Answer:
A. The bacteria are breaking down sugars in the absence of oxygen.
Explanation:
Bacteria are microscopic single-celled organims. These organisms may produce their own source of chemical energy, or consume and absorb chemical energy made by producers.
They break down chemical energy like glucose through lactic acid fermentation in their cytoplasm, without the presence of oxygen.
In Glycolysis:
2 molecules of ATP are used to break up glucose into 2 molecules of pyruvate, 4 ATP and 2 electron carrying NADH molecules. Since 2 ATP are used, a net 2ATP are produced by this process.
Then, pyruvate is converted to lactic acid, producing 2 NAD+, used as electron carriers.
Answer:
A) AUG GAG GGU
B) N-MET-GLU-GLY-HIS
C) 10 amino acids
D) The stop codon here is UAA, instead of UAG. If a tryptophane residue is added where the stop codon is, the GEN polypeptide would be longer
Explanation:
First we must obtain the the complementary chain or mRNA
A) AUG start codon is complementary to DNA TAC triplet, and GAG is complementary to CTC, and GGU to CCA (running from left to right towards 5'DNA direction (AAGCGGATACCTCCCAGTAGATGCTCCGGGGCTAGTGATTTCGAAC-5)
B) TAC corresponds to Methionine, GAG to Glutamate, GGU to Glycine, and CAU to Histidine
C) UAA is the stop codon, that is complementary where the DNA triplet is ATT. From AUG to UAA, we can have 10 amino acids transcribed
D) If a trp is included where the stop codon is UAA, then the mRNA would be longer with a consequent traduction of a longer GEN polypeptide
Answer:
The new drug will not be effective against the treatment of cancer.
Explanation:
The drug will not be effective against the cancer treatment because the drug will inhibit the dimerization of receptor tyrosine kinase (RTK) but as the Ras has undergone mutation due to replacement of one amino acid, it will be constitutively expressed and the drug is not effective in stopping the Ras functioning.
Thus, Gefitinib cannot work against the cancerous cell as it is not potent to stop the functioning of mutant Ras protein.
